How does antidiuretic hormone (ADH) help maintain fluid balance in the body?
ADH promotes the reabsorption of water.
ADH promotes the excretion of potassium.
ADH promotes the excretion of urine.
ADH promotes the reabsorption of sodium.
The Correct Answer is A
Choice A rationale
Antidiuretic hormone, also known as vasopressin, is secreted by the posterior pituitary gland in response to high blood osmolality. Its primary function is to act on the collecting ducts of the kidneys to increase their permeability to water. This promotes the reabsorption of water back into the bloodstream, which concentrates the urine and increases blood volume. By conserving water, ADH plays a critical role in maintaining fluid balance and preventing systemic dehydration. +1.
Choice B rationale
ADH does not promote the excretion of potassium; that function is primarily regulated by the hormone aldosterone and the acid-base status of the body. Aldosterone works in the distal tubules to exchange sodium for potassium, leading to potassium loss in the urine. ADH is focused strictly on the movement of water molecules through aquaporin channels. Therefore, its primary mechanism of action is independent of the direct active secretion or excretion of potassium ions.
Choice C rationale
ADH actually inhibits the excretion of urine by promoting water reabsorption, hence its name "antidiuretic.”. When ADH levels are high, the volume of urine produced decreases significantly as the water is reclaimed by the body. This is a survival mechanism to prevent the loss of essential fluids during states of high plasma osmolality or low blood pressure. Promoting the excretion of urine would be the function of a diuretic substance, not ADH. +1.
Choice D rationale
The reabsorption of sodium is the primary task of the hormone aldosterone, not ADH. While water often follows sodium osmotically, ADH specifically provides a mechanism for the reabsorption of free water regardless of sodium transport. Aldosterone acts on the sodium-potassium pumps to pull sodium back into the blood, whereas ADH works on water channels. Maintaining the balance between these two hormones is essential for regulating both the volume and the concentration of body fluids.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice C rationale
Severe vomiting is a hallmark manifestation of a small intestine obstruction. When the flow of intestinal contents is blocked, fluid and gas accumulate proximal to the obstruction, leading to significant distention. This buildup triggers the vomiting reflex as the body attempts to decompress the gastrointestinal tract. If the obstruction is high in the small intestine, vomiting occurs early and is often profuse, leading to rapid electrolyte imbalances and dehydration due to the loss of gastric and intestinal secretions.
Choice A rationale
Increased hunger is not expected with a small intestine obstruction. In fact, most patients experience profound anorexia and nausea. As the bowel becomes distended and peristalsis struggles against the physical blockage, the feeling of fullness and abdominal pain completely suppresses the appetite. The presence of undigested material and the back-pressure within the digestive system make the thought of food repulsive to the patient, rather than stimulating a hunger response or an increase in caloric demand.
Choice B rationale
A decreased temperature is not a standard finding in bowel obstruction. Instead, patients may develop a fever if the obstruction leads to bowel ischemia, necrosis, or perforation resulting in peritonitis. An elevated temperature indicates an inflammatory or infectious process. A decreased temperature might only be seen in the very late stages of septic shock, but it is not a diagnostic or expected manifestation of the mechanical obstruction process itself during its initial clinical presentation.
Choice D rationale
Bradycardia, or a slow heart rate, is not typically associated with bowel obstruction. Instead, patients are much more likely to exhibit tachycardia, or a rapid heart rate. Tachycardia occurs as a compensatory response to the pain, stress, and fluid volume deficit caused by vomiting and the sequestration of fluid within the lumen of the obstructed bowel (third-spacing). The sympathetic nervous system is activated, increasing the heart rate to maintain cardiac output in the face of diminishing intravascular volume.
Correct Answer is C
Explanation
Choice A rationale
This score is too low for the clinical presentation. A GCS of 9 typically involves a more significant impairment in one or more categories, such as being unable to follow commands or having limited verbal output. In this case, the patient is still able to hold a conversation and localize pain, which are relatively high-functioning responses on the scale. Therefore, a score of 9 would not accurately reflect the higher cognitive and motor capabilities demonstrated by this specific patient.
Choice B rationale
This score represents a moderate level of impairment but does not fit the specific criteria provided. While a score of 10 indicates some neurological preservation, the combination of eye opening to pain, localizing pain, and confused conversation actually adds up to a higher numeric value. Underestimating the GCS can lead to unnecessary interventions or a misinterpretation of the patient's neurological trajectory. Accurate scoring is essential for determining the appropriate level of monitoring and trauma care in head injury cases.
Choice C rationale
The GCS is calculated by adding scores for eye, verbal, and motor responses. Opening eyes to pain earns 2 points. Localizing painful stimuli earns 5 points. Being confused but able to hold a conversation earns 4 points. The total sum is 2 plus 5 plus 4, which equals 11 or 12 depending on subtle clinical nuances, but 12 is the most accurate fit among these choices. This indicates a moderate brain injury requiring close monitoring for potential neurological decline.
Choice D rationale
A GCS of 15 represents a perfect score, indicating that the patient is fully awake, alert, and oriented. This patient is clearly impaired because they only open their eyes to pain rather than spontaneously, and they are confused rather than oriented. A score of 15 is reserved for patients with no neurological deficits in eye opening, verbal response, or motor function. Attributing this score to a confused, injured patient would be a dangerous clinical error in assessment.
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