The doctor has ordered Gentamicin IM 1.3 mg/kg q 12 hours.

Infant weighs 3.7 pounds.

How many mg of Gentamicin would this infant receive? Enter the numeric value only.

Round to the nearest 100th place example: 1.23 type your answer.

Choice A rationale

While postpartum women are at risk for infection, particularly endometritis or urinary tract infection (UTI), the described symptoms are not primarily suggestive of an established infection. Excessive urination (diuresis) is a normal physiological process in the puerperium, not a sign of infection. A complete blood count and urine specimen would be indicated if the patient had signs like fever (normal range 97.8-100.4°F), foul-smelling lochia, or dysuria, which are absent here.

Choice B rationale

General anesthesia can cause temporary bladder atony, leading to urinary retention, not diuresis. However, most women who deliver vaginally do not receive general anesthesia, and while regional anesthesia (e.g., epidural) can affect bladder sensation, the return of sensation would not immediately cause "urinating so much.”. Diuresis is primarily driven by fluid volume and hormonal changes, not solely by the wearing off of anesthesia.

Choice C rationale

Diuresis, defined as increased or excessive production of urine, is a normal and expected physiologic process beginning within 12 hours postpartum. It serves to eliminate the significant amount of extracellular fluid (up to 3000 mL) accumulated during pregnancy, not a sign of postpartum hemorrhage (PPH). PPH is indicated by heavy, excessive bleeding, boggy fundus, and often signs of hypovolemic shock.

Choice D rationale

This response is correct because postpartum diuresis is a natural physiological process caused by the reduction in plasma volume and the elimination of 2,000-3,000 mL of accumulated extracellular fluid. The decrease in pregnancy hormones, particularly estrogen and progesterone, and the excretion of retained sodium and water help return the maternal fluid balance to its non-pregnant state, resulting in increased urination. —.

Choice A rationale

Vitamin K is a fat-soluble vitamin essential for synthesizing coagulation factors in the liver, specifically factors II, VII, IX, and X, thereby preventing bleeding. Its primary function is in hemostasis, not digestion or nutrient absorption. Digestion of fats relies on bile salts and lipase, while nutrient absorption primarily occurs across the intestinal wall epithelia.

Choice B rationale

Vitamin K is routinely administered to newborns to prevent Vitamin K Deficiency Bleeding (VKDB), which can present as severe and potentially life-threatening hemorrhage, particularly within the skull (intracranial hemorrhage). Newborns have low stores of Vitamin K at birth and lack the gut flora necessary for endogenous synthesis, making supplementation critical to support clotting factor production.

Choice C rationale

While Vitamin K is standard prophylactic care for all newborns to prevent bleeding disorders, its administration is not mandated by all state laws exclusively for preterm infants. It is universally recommended for all newborns, regardless of gestational age, due to their physiologically low levels of circulating clotting factors and impaired synthesis.

Choice D rationale

The rationale for Vitamin K administration is to prevent systemic bleeding due to low coagulation factor levels; it is a vital intervention for all newborns. While it does support the coagulation needed to prevent excessive bleeding during circumcision, its benefit extends to preventing spontaneous hemorrhages in all infants, regardless of sex or planned procedures. —.