The protein coat surrounding viral genetic material is the:
Capsid
Cell wall
Cytoplasm
The Correct Answer is A
A. Capsid: The capsid is the protein shell that encloses and protects the viral nucleic acid, which can be either DNA or RNA. It is composed of repeating protein subunits called capsomeres, which provide structural stability and facilitate attachment to host cells. The capsid plays a key role in viral infectivity and immune system recognition.
B. Cell wall: A cell wall is a rigid structure found in bacteria, fungi, and plants that provides shape and protection to the cell. Viruses do not have cell walls, as they are acellular entities and rely on host cells for replication.
C. Cytoplasm: Cytoplasm is the gel-like substance inside cells that contains organelles and metabolic machinery. Viruses lack cytoplasm entirely, as they are not true cells and cannot carry out independent metabolic processes.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A. Chitin: Fungal cell walls are primarily composed of chitin, a long-chain polymer of N-acetylglucosamine. Chitin provides structural strength and rigidity to the fungal cell wall, allowing the organism to maintain shape and resist osmotic pressure. Its presence distinguishes fungi from bacteria and makes it a target for antifungal therapies.
B. Peptidoglycan: Peptidoglycan is a key structural component of bacterial cell walls, particularly in Gram-positive and Gram-negative bacteria. It provides rigidity and protects against osmotic lysis but is absent in fungal cells, so it is not responsible for their structural integrity.
C. Lipopolysaccharide: Lipopolysaccharides (LPS) are molecules found in the outer membrane of Gram-negative bacteria. They contribute to bacterial virulence and can trigger immune responses in humans. LPS is not found in fungi and does not play a role in fungal infections.
Correct Answer is D
Explanation
A. Nonsense and deletion: A nonsense mutation occurs when a codon that normally codes for an amino acid is changed into a stop codon, causing premature termination of protein synthesis. Although deletions can cause frameshifts, nonsense mutations themselves do not alter the reading frame. Therefore, this combination does not consistently produce frameshift mutations.
B. Missense and insertion: A missense mutation results from a single nucleotide substitution that changes one amino acid in the protein sequence. This alters protein structure but does not shift the reading frame. While insertions can cause frameshifts, the combination listed does not accurately represent the mechanisms responsible for frameshift mutations.
C. Missense and nonsense: Both missense and nonsense mutations are types of point mutations caused by single base substitutions. These mutations affect the identity of a codon or create a premature stop codon but do not alter the grouping of codons into triplets. As a result, they do not shift the reading frame of the genetic code.
D. Deletion and insertion: Frameshift mutations occur when nucleotides are inserted into or deleted from the DNA sequence in numbers not divisible by three. Because the genetic code is read in triplets (codons), such changes shift the reading frame downstream of the mutation. This alters all subsequent codons and often results in a drastically altered or nonfunctional protein.
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