What arterial blood gas result is expected in a person experiencing a panic attack?
pH 7.34, PaCO2 45 mm Hg, HCO3 21 mEq/L.
pH 7.32, PaCO2 49 mm Hg, HCO3 26 mEq/L.
pH 7.46, PaCO2 42 mm Hg, HCO3 28 mEq/L.
pH 7.49, PaCO2 28 mm Hg, HCO3 24 mEq/L.
The Correct Answer is D
Choice D rationale
A panic attack typically involves hyperventilation, where the person breathes rapidly and deeply. This causes them to blow off excessive amounts of carbon dioxide (CO2). According to the arterial blood gas values, a low PaCO2 (less than 35 mmHg) leads to an increase in pH (greater than 7.45), resulting in respiratory alkalosis. Choice D shows a pH of 7.49 and a PaCO2 of 28 mmHg, with a normal HCO3 of 24 mEq/L, which perfectly illustrates the acute respiratory alkalosis expected during hyperventilation.
Choice A rationale
Choice A represents a state of mild metabolic acidosis or a near-normal result, with a pH of 7.34 and a PaCO2 of 45 mmHg. In a panic attack, the primary change is a decrease in CO2 due to rapid breathing, not an increase or a drop in pH. This profile suggests the body is retaining too much acid or losing base, which is the physiological opposite of what happens when a person hyperventilates during an episode of intense acute anxiety or panic.
Choice B rationale
Choice B reflects respiratory acidosis, indicated by a low pH of 7.32 and an elevated PaCO2 of 49 mmHg. This occurs in conditions where ventilation is inadequate, such as chronic obstructive pulmonary disease or respiratory depression, leading to the retention of carbon dioxide. In a panic attack, the person is over-ventilating, not under-ventilating, so the PaCO2 should be low and the pH should be high, making this result incorrect for the described clinical scenario.
Choice C rationale
Choice C shows a pH of 7.46 and an HCO3 of 28 mEq/L with a normal PaCO2. This indicates metabolic alkalosis. While the pH is high, the cause is an excess of bicarbonate rather than a deficit of carbon dioxide. In a panic attack, the alkalosis is driven by the respiratory system (low PaCO2) rather than a metabolic gain of base. Therefore, this result does not align with the rapid breathing and CO2 loss characteristic of a panic-induced hyperventilation event. .
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A rationale
Infants have a much higher body surface area to volume ratio compared to adults. This physiological characteristic means that a larger proportion of their total body water is in contact with the environment, allowing for rapid fluid loss through the skin via evaporation. Because infants have more skin surface relative to their small body mass, even minor increases in environmental temperature or respiratory rate can lead to significant fluid deficits, making them scientifically more prone to rapid dehydration.
Choice B rationale
Subcutaneous fat actually serves as an insulating layer and does not increase the risk of dehydration. In fact, adipose tissue has a lower water content than muscle tissue, but its presence does not predispose an infant to fluid loss. Infants typically have a higher percentage of total body water, approximately 75 percent, compared to adults. The risk of dehydration is related to how that water is distributed and lost, not the amount of subcutaneous fat present on the infant's body.
Choice C rationale
Forgetfulness regarding fluid intake is a behavioral risk factor often associated with the elderly or older children, but it is not applicable to infants. Infants rely entirely on caregivers for fluid administration and cannot consciously choose or forget to drink. Their risk for dehydration is primarily driven by physiological and anatomical factors, such as their high metabolic rate and immature renal function, which limit their ability to concentrate urine and conserve water when fluid intake is insufficient.
Choice D rationale
The sweat mechanism in infants is actually immature and less efficient than in adults. Infants do not sweat as effectively to dissipate heat, which puts them at a higher risk for overheating rather than dehydration specifically through sweating. While they do lose fluid through the skin, the primary issue is the high surface area to volume ratio and a higher respiratory rate, which increases insensible water loss, rather than an overactive or increased sweat mechanism compared to older individuals.
Correct Answer is B
Explanation
Choice A rationale
Metabolic alkalosis occurs when the pH is above 7.45 and the bicarbonate level is above 26 mEq/L. This condition is typically caused by a loss of hydrogen ions or an excess of bicarbonate. In this clinical scenario, the pH is 7.30 and the bicarbonate is 24 mEq/L, which is within the normal range of 22 to 28 mEq/L. Therefore, the imbalance cannot be classified as metabolic alkalosis based on these laboratory values.
Choice B rationale
Respiratory acidosis is characterized by a pH below 7.35 and a PaCO2 above 45 mmHg. The normal range for pH is 7.35 to 7.45, and for PaCO2 is 35 to 45 mmHg. In this case, the pH of 7.30 indicates acidemia, and the elevated PaCO2 of 50 mmHg identifies the respiratory system as the cause. The normal bicarbonate level suggests that renal compensation has not yet occurred, confirming an acute respiratory acid-base imbalance.
Choice C rationale
Respiratory alkalosis involves a pH greater than 7.45 and a PaCO2 less than 35 mmHg. This state is generally caused by hyperventilation, which leads to the excessive elimination of carbon dioxide from the lungs. Since the patient in this scenario has a low pH of 7.30 and a high carbon dioxide level of 50 mmHg, the laboratory data is the direct opposite of what is required for a diagnosis of respiratory alkalosis.
Choice D rationale
Metabolic acidosis is defined by a pH below 7.35 and a bicarbonate level below 22 mEq/L. While the pH of 7.30 in this scenario indicates acidosis, the bicarbonate level of 24 mEq/L is perfectly within the standard reference range of 22 to 28 mEq/L. Because the primary abnormality is found in the PaCO2 level rather than the bicarbonate level, the source of the acid-base disturbance is respiratory and not metabolic in origin.
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