What is the best position the nurse can place the patient with increased intracranial pressure in?
Elevate the head of the bed to 10 degrees and keep the head in a natural position.
Elevate the head of the bed to 30 degrees and keep the head in a neutral midline position.
Keep the head of the bed flat with the head in a neutral midline position.
Keep the head of the bed flat with the head in a natural position.
The Correct Answer is B
Rationale:
A. Elevating the head of the bed to 10 degrees is incorrect because 10 degrees is usually insufficient to promote optimal venous drainage from the brain. Keeping the head in a natural (turned) position can also impair venous return, potentially increasing ICP.
B. Elevating the head of the bed to 30 degrees with the head in a neutral midline position is correct. This position promotes venous drainage from the brain, reduces intracranial pressure, and maintains cerebral perfusion. Neutral alignment prevents kinking of the jugular veins, which could impede venous outflow. Elevating the head too much (>45 degrees) can decrease cerebral perfusion, while flat positioning can worsen ICP.
C. Keeping the head of the bed flat, even with the head in a neutral midline position, is incorrect because flat positioning increases venous congestion in the brain, which can raise ICP.
D. Keeping the head of the bed flat with the head in a natural (turned) position is incorrect for the same reason as C and adds risk of impaired venous drainage due to rotation of the neck, which can further elevate ICP.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["A","B","C","E"]
Explanation
Rationale:
A. Deep, rapid breathing (Kussmaul respirations) is a compensatory response to metabolic acidosis caused by excess ketone accumulation. By breathing faster and deeper, the patient exhales more carbon dioxide, which helps reduce acid levels in the blood. This finding indicates severe acidosis and is a classic, high-priority sign in DKA.
B. Decreased urine output occurs as DKA progresses due to severe dehydration. Initially, hyperglycemia causes osmotic diuresis, leading to excessive urination. Over time, profound fluid loss results in hypovolemia, decreased renal perfusion, and ultimately reduced urine output, making this an expected assessment finding on admission.
C. Elevated blood glucose level is a defining characteristic of DKA. Insulin deficiency prevents glucose from entering the cells, causing blood glucose to rise significantly (often >250 mg/dL). This hyperglycemia contributes to dehydration, electrolyte imbalances, and worsening acidosis.
D. Increase in pH is incorrect because DKA causes metabolic acidosis, leading to a decreased pH (typically <7.35). The presence of ketone bodies lowers the blood pH, which is responsible for many of the patient’s clinical manifestations.
E. Acetone (fruity) breath results from the accumulation of ketones, particularly acetone, which is exhaled through the lungs. This distinct odor is a classic assessment finding and helps differentiate DKA from other hyperglycemic conditions such as hyperosmolar hyperglycemic syndrome (HHS).
Correct Answer is D
Explanation
Rationale:
A. Progressive is incorrect because this stage of shock occurs after the compensatory stage when the body’s mechanisms begin to fail, leading to tissue hypoperfusion, worsening acidosis, and organ dysfunction. However, interventions at this stage can still potentially reverse damage if promptly addressed.
B. Compensatory is incorrect because, in this early stage, the body activates mechanisms such as increased heart rate, vasoconstriction, and respiratory changes to maintain perfusion. Tissue perfusion is still adequate, and damage is usually reversible.
C. Initial is incorrect because this is the very beginning of shock, often asymptomatic, where cellular changes begin but clinical signs may be minimal. Early interventions are highly effective at this stage.
D. Refractory is correct because this is the final stage of shock, sometimes called irreversible shock. In this stage, prolonged hypoperfusion and cellular injury lead to widespread organ failure. Even with aggressive treatment, the damage is often irreversible, and death is likely.
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