Which assessment finding is most indicative of autonomic dysreflexia in a client with a spinal cord injury?
Warm dry skin
Severe hypertension and headache
Slow breathing
Low blood pressure
The Correct Answer is B
In autonomic dysreflexia, a life-threatening sympathetic hyperreflexic response occurs in spinal cord injury above T6 due to noxious stimuli below lesion, producing sudden hypertension, severe headache, and bradycardia episodes clinically.
Rationale:
A. Warm dry skin suggests intact sympathetic thermoregulation and is inconsistent with autonomic dysreflexia. In dysreflexia, cutaneous flushing and diaphoresis occur above lesion. The finding contradicts sympathetic overactivity. Therefore this option does not reflect hemodynamic crisis seen in spinal cord injury.
B. Severe hypertension and headache are hallmark manifestations of autonomic dysreflexia resulting from uninhibited sympathetic discharge. The blood pressure elevation is sudden and severe. Headache reflects intracranial pressure changes and cerebral vasodilation secondary to extreme hypertension in spinal cord injury.
C. Slow breathing is not a defining feature of autonomic dysreflexia, which primarily involves sympathetic overactivity above lesion level. Respiratory rate is typically normal unless complications arise. Bradycardia rather than tachypnea is more characteristic of the autonomic imbalance.
D. Low blood pressure is inconsistent with autonomic dysreflexia and typically suggests hypovolemia or neurogenic shock. In autonomic dysreflexia, sympathetic discharge causes marked vasoconstriction above lesion. Resulting physiology elevates systemic vascular resistance and blood pressure. Therefore hypotension is not expected finding.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
The Glasgow Coma Scale (GCS) is a standardized neurological tool used to assess level of consciousness, evaluating eye opening, verbal response, and motor response. A declining score reflects worsening cerebral function, often due to increased intracranial pressure or evolving brain injury.
Rationale:
A. A decrease from 14 to 10 is not a normal finding. Normal neurological status is reflected by stable or improving GCS scores. A drop indicates deterioration in cortical or brainstem function, often linked to hypoxia, hemorrhage, or increased intracranial pressure progression.
B. A declining GCS score does not represent a stable condition. Stability requires no change or minimal fluctuation in neurological status. A drop of 4 points is clinically significant and suggests ongoing neurological compromise requiring immediate reassessment and possible escalation of care.
C. A reduction in GCS from 14 to 10 is not an improvement. Improvement would involve an increasing score indicating better eye, verbal, and motor responses. The observed decrease reflects reduced consciousness and impaired neurological responsiveness rather than recovery.
D. A falling GCS score indicates worsening neurological status. This change suggests progressive cerebral dysfunction potentially due to intracranial bleeding, hypoxia, or brain edema. It is a critical deterioration requiring urgent neurological evaluation and possible emergency intervention to prevent further decline.
Correct Answer is D
Explanation
Atelectasis is alveolar collapse caused by impaired ventilation, retained secretions, decreased lung expansion, and ineffective cough mechanisms. Clients with spinal cord injury commonly develop respiratory compromise due to diaphragmatic weakness, reduced chest expansion, mucus retention, and diminished pulmonary clearance capacity.
Rationale:
A. Chest muscle strengthening may improve long-term respiratory endurance but does not directly prevent alveolar collapse in acute pulmonary care. Atelectasis prevention primarily requires airway clearance and lung expansion techniques. Ineffective secretion removal promotes mucus obstruction and reduced alveolar ventilation in immobilized clients.
B. Panting breaths produce shallow respirations that inadequately expand distal alveoli and do not effectively mobilize pulmonary secretions. Atelectasis prevention requires sustained inspiratory effort and forceful secretion clearance techniques. Shallow breathing worsens alveolar collapse and contributes to impaired gas exchange within dependent lung regions.
C. Excessive rest promotes immobility, shallow respirations, and secretion stasis, increasing pulmonary complication risk after spinal cord injury. Respiratory exercises and mobilization improve ventilation and secretion clearance. Prolonged inactivity contributes to hypoventilation and accumulation of retained bronchial secretions within the lower respiratory tract.
D. Deep coughing promotes maximal lung expansion and mobilizes retained respiratory secretions, making it essential for atelectasis prevention. Forceful coughing increases airway clearance and improves oxygenation in clients with weakened respiratory musculature. Effective pulmonary hygiene reduces secretion retention and prevents progressive alveolar collapse.
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