You're providing care to a patient who is being treated for aspiration pneumonia. The patient is on a 100% non-rebreather mask. Which finding below is a hallmark sign and symptom that the patient is developing acute respiratory distress syndrome (ARDS)?
The patient is experiencing bradypnea.
The patient is tired and confused.
The patient's PaO2 remains at 45 mmHg.
The patient's blood pressure is 180/96.
The Correct Answer is C
Rationale:
A. The patient is experiencing bradypnea is incorrect because ARDS is typically associated with tachypnea, not bradypnea. Patients initially respond to hypoxemia by increasing their respiratory rate to improve oxygenation.
B. The patient is tired and confused is incorrect because while altered mental status can occur due to hypoxemia, it is a secondary symptom, not the hallmark diagnostic feature of ARDS.
C. The patient's PaO2 remains at 45 mmHg is correct. A hallmark of ARDS is severe hypoxemia that persists despite 100% oxygen delivery. In ARDS, inflammation and alveolar damage lead to non-cardiogenic pulmonary edema, reducing gas exchange. This refractory hypoxemia is a key diagnostic criterion for ARDS and distinguishes it from other causes of respiratory distress.
D. The patient's blood pressure is 180/96 is incorrect because ARDS is primarily a pulmonary disorder, not a hypertensive emergency. Blood pressure changes may occur secondary to stress or sepsis, but they are not hallmark signs of ARDS.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["B","E"]
Explanation
Rationale:
A. Anxiety and fear usually increase respiratory rate and depth (hyperventilation). Hyperventilation blows off CO2, which can lead to respiratory alkalosis, not acidosis. Therefore, anxiety is not a predisposing factor for respiratory acidosis.
B. CNS depression from conditions such as stroke, brain injury, sedation, or neurologic disease can suppress the respiratory drive, leading to hypoventilation. Hypoventilation results in CO2 retention, which increases PaCO2 and lowers pH, producing respiratory acidosis.
C. DKA causes metabolic acidosis due to the accumulation of ketone bodies. The patient often develops Kussmaul respirations (deep, rapid breathing) to blow off CO2 as a compensatory mechanism, which is opposite of respiratory acidosis.
D. NG suctioning removes gastric acid, which can lead to loss of hydrogen ions, causing metabolic alkalosis, not respiratory acidosis. It does not directly affect CO2 elimination or the respiratory system.
E. Sedatives such as benzodiazepines, opioids, or barbiturates depress the CNS, including the respiratory centers in the medulla. This leads to hypoventilation, decreased alveolar ventilation, and CO2 retention, resulting in respiratory acidosis.
Correct Answer is C
Explanation
Rationale:
A. This oxygen saturation is within normal limits (95–100%). While age may slightly reduce baseline oxygenation, 96% is acceptable, and the patient is stable. There is no immediate threat to respiratory function, so this patient does not require urgent attention.
B. A saturation of 94% is slightly below normal but generally considered mild hypoxemia. The patient is likely compensating and may not require immediate intervention. Continuous monitoring is appropriate, but urgent evaluation is not required unless symptoms develop.
C. This patient is hypoxemic despite supplemental oxygen, which is more serious than a patient with normal or slightly low oxygen on room air. Normally, PaO2 should rise above 80 mm Hg with supplemental oxygen. Failure to achieve adequate oxygenation on 2 L/min indicates compromised gas exchange, possibly from pneumonia, ARDS, pulmonary embolism, or worsening respiratory failure. This patient is at highest risk for rapid deterioration, making them the priority. Immediate assessment, titration of oxygen, or escalation of care may be required.
D. PaO2 of 94 mm Hg is within normal limits (80–100 mm Hg). This patient is stable and does not require urgent intervention.
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