Your patient had emergency surgery to evacuate a subdural hematoma yesterday and appears to be doing well.
However, during your shift, you have suddenly had to empty the patient's Foley bag 4 times in the last 6 hours. You suspect your patient has developed what complication?

Choice A rationale

The patient's initial unconsciousness, followed by a lucid interval and then declining consciousness (appearing to fall asleep, unable to verbally reply), is a classic presentation of an epidural hematoma. This condition requires immediate medical attention as expanding hematomas can lead to brain herniation. Notifying the MD and preparing for a burr-hole procedure to relieve intracranial pressure is the highest priority to prevent irreversible brain damage.

Choice B rationale

While pain management is important, it is not the priority in a patient with a rapidly deteriorating neurological status. Administering pain medication without addressing the underlying intracranial pathology could mask critical neurological signs and delay life-saving interventions. The immediate concern is the potential for brain compression, not comfort.

Choice C rationale

Placing the patient on a nasal cannula at 2 Lpm for a patient with deteriorating consciousness is insufficient and potentially inappropriate. If the patient's respiratory drive is compromised due to increased intracranial pressure, more aggressive airway management, potentially intubation, might be required. Oxygenation should be assessed and managed, but it is not the initial priority without evaluating the airway and breathing comprehensively in a declining patient.

Choice D rationale

Obtaining a stat EKG is not the priority action in a patient presenting with acute neurological deterioration following head trauma. While cardiac function is important, the immediate threat to life in this scenario is neurological compromise due to potential intracranial bleeding and rising intracranial pressure. An EKG would be a secondary assessment after stabilizing the primary neurological issue.

Choice A rationale

Defecation, while it can occur during severe neurological events due to autonomic dysregulation, is a non-specific sign and not a primary or direct indicator of brain herniation. Brain herniation primarily affects vital centers and cranial nerves, leading to more direct neurological compromise.

Choice B rationale

Pinpoint pupils are typically associated with pontine lesions or opioid overdose due to parasympathetic overactivity. In contrast, brain herniation, especially uncal herniation, often causes ipsilateral pupil dilation due to compression of the oculomotor nerve (cranial nerve III), leading to parasympathetic blockade.

Choice C rationale

Tachycardia, an increased heart rate, can be a non-specific response to stress, pain, or hypovolemia. In the context of brain herniation, as intracranial pressure rises and compresses the brainstem, bradycardia (slowing of the heart rate) is a more characteristic finding due to the Cushing reflex, not tachycardia.

Choice D rationale

Bilateral dilated pupils, especially when fixed and non-reactive to light, are a critical and often late sign of severe brain herniation, indicating significant brainstem compression and widespread damage, particularly to the midbrain's oculomotor nerve nuclei or their pathways. This signifies severe cerebral anoxia or irreversible brain injury.