A 32-year-old male is admitted to the ICU following a severe motor vehicle accident. He has a Glasgow Coma Scale (GCS) score of 7. Initial CT scan shows a large subdural hematoma with midline shift. His vital signs are: BP 180/100 mmHg, HR 50 bpm, RR B breaths/min, SpO2 96% on mechanical ventilation. The nurse notes that his right pupil has suddenly become dilated and nonreactive. What is going on?
Patient is likely having a brain herniation
Patient is likely having an ischemic stroke
Patient is severely dehydrated
Patient has uncontrolled hypertension
The Correct Answer is A
A. Patient is likely having a brain herniation is correct because the combination of sudden unilateral pupillary dilation, hypertension, bradycardia, and abnormal respirations in the context of a large subdural hematoma and midline shift is indicative of increased intracranial pressure (IICP) leading to brain herniation. This is a neurological emergency. The fixed and dilated pupil suggests pressure on the oculomotor nerve (cranial nerve III) due to uncal herniation. The vital signs reflect Cushing’s triad: hypertension (widened pulse pressure), bradycardia, and irregular respirations, which are compensatory mechanisms to maintain cerebral perfusion. Immediate intervention is critical to prevent death.
B. Patient is likely having an ischemic stroke is incorrect because while ischemic strokes can cause neurological deficits, they do not typically produce the acute pupillary changes, midline shift, and Cushing’s triad seen in this patient. Additionally, the history of trauma and large subdural hematoma supports herniation rather than primary stroke.
C. Patient is severely dehydrated is incorrect because dehydration would typically cause tachycardia and hypotension, not bradycardia and hypertension. Dehydration also would not cause pupillary dilation or midline shift on imaging.
D. Patient has uncontrolled hypertension is incorrect because although the patient is hypertensive, the acute neurological signs (fixed dilated pupil, GCS 7, midline shift) are secondary to IICP and herniation, not primary hypertension. The elevated BP is part of Cushing’s response, not the underlying cause.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Normal ICP is incorrect because normal intracranial pressure ranges from 5 to 15 mmHgin adults. Values above 20 mmHg are considered elevated, and sustained ICP above 20–25 mmHg is critically highand requires urgent intervention.
B. Mildly elevated ICP is incorrect because ICP of 28 mmHg exceeds mild elevation, which is generally considered 20–25 mmHg. This patient’s ICP is above that range and represents a dangerous level.
C. Critically elevated ICP is correct. An ICP of 28 mmHg indicates critically elevated intracranial pressure, especially when accompanied by low CPP (normal 60–70 mmHg)and hypercapnia (PaCO2 60 mmHg). Hypercapnia causes cerebral vasodilation, further increasing ICP and worsening cerebral perfusion. Immediate interventions are required to reduce ICP, optimize CPP, and prevent secondary brain injury.
Correct Answer is B
Explanation
A. Increase hydrogen ions in blood is incorrect because metabolic acidosis (accumulation of hydrogen ions) can occur in CKD but does not directly cause anemia. It contributes more to bone demineralization and other metabolic disturbances rather than affecting red blood cell production.
B. Decreased erythropoietin production is correct because the kidneys produce erythropoietin, a hormone that stimulates the bone marrow to produce red blood cells. In CKD, damaged kidneys cannot produce sufficient erythropoietin, leading to decreased red blood cell production and anemia. This is the most common cause of anemia in CKD and often requires treatment with recombinant erythropoietin or iron supplementation.
C. Change in white blood cell function is incorrect because alterations in white blood cells may occur with CKD-related immune dysfunction, but they do not cause anemia. Anemia is specifically related to red blood cell production and lifespan.
D. Hyperinsulinemia is incorrect because elevated insulin levels are not directly linked to anemia in CKD. Insulin abnormalities are more commonly associated with metabolic syndrome or diabetes, which may coexist with CKD but are not primary causes of anemia in this condition.
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