A client who has uncontrolled diabetes mellitus visits a clinic. Client reports numbness and tingling in the feet which keeps them up at night. The nurse explains this condition as a nerve disorder called peripheral neuropathy. Which of the following statements by the client indicates an understanding of what causes this condition?
"The older I get, there is less blood flowing to my feet."
"The elevated blood sugar from my diabetes can cause underlying nerve damage."
"The nerves just go to sleep when I lie down because no message gets from my brain to the spinal cord."
"The nerve damage may occur for unknown reasons in any individual."
The Correct Answer is B
Diabetic peripheral neuropathy is a chronic microvascular and metabolic complication of diabetes mellitus caused by prolonged hyperglycemia leading to axonal degeneration, demyelination, and ischemic injury of peripheral nerves. It commonly presents with distal symmetric sensory loss, burning pain, and paresthesia, especially in the lower extremities.
Rationale:
A. Age-related reduced perfusion may contribute to vascular insufficiency but does not explain diabetic neuropathy pathophysiology. Peripheral neuropathy in diabetes is primarily driven by metabolic and microvascular injury rather than physiologic aging changes affecting distal limb circulation alone.
B. Chronic hyperglycemia leads to non-enzymatic glycation of proteins, oxidative stress, and microvascular ischemia causing nerve damage in diabetic peripheral neuropathy. Sustained elevated glucose disrupts Schwann cell function and axonal conduction, producing sensory symptoms such as burning, tingling, and nocturnal pain.
C. Peripheral neuropathy is not caused by temporary functional sleep of nerves or interruption between brain and spinal cord signaling. It results from structural nerve injury due to metabolic and ischemic mechanisms rather than reversible conduction block or positional neural inactivity.
D. Diabetic neuropathy is not idiopathic in uncontrolled diabetes mellitus. It has a well-established pathophysiology involving chronic hyperglycemia-induced microvascular injury and metabolic toxicity. Although some neuropathies are idiopathic, diabetic neuropathy has a clearly defined causal mechanism.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
In critically ill patients, the ABCDEF bundle is an evidence-based ICU protocol targeting pain management, sedation optimization, delirium prevention, early mobility, and family engagement to improve outcomes and reduce ventilator days and long-term cognitive dysfunction in intensive care.
Rationale:
A. Activity is part of later components in the ABCDEF bundle focusing on early mobilization and physical rehabilitation. It is not the first priority. While important for preventing ICU-acquired weakness, it does not represent the initial A step which centers on pain assessment and control in critically ill clients.
B. Assess pain represents the correct A component of the ABCDEF bundle. The initial focus is pain assessment using validated scales to identify discomfort early. Effective analgesia-first strategies reduce agitation, improve ventilator synchrony, and decrease unnecessary sedation exposure in critically ill patients. This is the correct intervention.
C. Alertness refers more closely to sedation level and neurological status monitoring rather than the A component. While important in ICU delirium prevention strategies, it is not the primary A step. The ABCDEF bundle prioritizes pain evaluation before assessing consciousness or sedation depth in critically ill clients.
D. Airway management is fundamental in critical care but belongs to primary ABC resuscitation principles, not the ABCDEF bundle structure. The bundle assumes airway is already secured. The A in this protocol specifically targets pain assessment and management rather than airway stabilization or respiratory support interventions.
Correct Answer is D
Explanation
The Glasgow Coma Scale (GCS) is a standardized neurological tool used to assess level of consciousness, evaluating eye opening, verbal response, and motor response. A declining score reflects worsening cerebral function, often due to increased intracranial pressure or evolving brain injury.
Rationale:
A. A decrease from 14 to 10 is not a normal finding. Normal neurological status is reflected by stable or improving GCS scores. A drop indicates deterioration in cortical or brainstem function, often linked to hypoxia, hemorrhage, or increased intracranial pressure progression.
B. A declining GCS score does not represent a stable condition. Stability requires no change or minimal fluctuation in neurological status. A drop of 4 points is clinically significant and suggests ongoing neurological compromise requiring immediate reassessment and possible escalation of care.
C. A reduction in GCS from 14 to 10 is not an improvement. Improvement would involve an increasing score indicating better eye, verbal, and motor responses. The observed decrease reflects reduced consciousness and impaired neurological responsiveness rather than recovery.
D. A falling GCS score indicates worsening neurological status. This change suggests progressive cerebral dysfunction potentially due to intracranial bleeding, hypoxia, or brain edema. It is a critical deterioration requiring urgent neurological evaluation and possible emergency intervention to prevent further decline.
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