A patient is prescribed 20 mg of Paroxetine daily. The available tablet strength is 10 mg. How many tablets should be administered?
3 tablets
4 tablets
1 tablet
2 tablets
The Correct Answer is D
Choice A reason: Three tablets (30 mg) exceed the prescribed 20 mg, risking Paroxetine overdose. This SSRI increases serotonin levels, and excessive dosing may cause serotonin syndrome, with symptoms like agitation or seizures. Scientifically, precise dosing is critical to avoid disrupting neurotransmitter balance, ensuring safe treatment for depression or anxiety.
Choice B reason: Four tablets (40 mg) significantly exceed the prescribed dose, increasing toxicity risk. Paroxetine’s serotonin-enhancing effects can lead to severe side effects, like serotonin syndrome or cardiovascular issues, if overdosed. Scientifically, maintaining therapeutic levels prevents adverse neurological effects, ensuring safe and effective management of mental health conditions.
Choice C reason: One tablet (10 mg) underdoses the patient, providing half the required 20 mg. Subtherapeutic Paroxetine levels fail to adequately increase serotonin, reducing efficacy for depression or anxiety. Scientifically, insufficient dosing may leave neural imbalances unaddressed, risking persistent symptoms and poor mental health outcomes.
Choice D reason: Two tablets (20 mg) match the prescribed dose, ensuring therapeutic serotonin levels for depression or anxiety management. Scientifically, accurate SSRI dosing optimizes neurotransmitter balance, stabilizing mood without risking toxicity. This supports effective treatment, reducing symptoms like low mood or anxiety through targeted neurochemical modulation.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A reason: Immediate psychiatric referral without engagement skips assessment, risking inappropriate care. PTSD involves amygdala-driven hyperarousal, requiring initial stabilization. Scientifically, bypassing patient interaction may delay addressing specific trauma triggers, leaving neurobiological stress responses, like cortisol spikes, unmanaged, hindering effective PTSD recovery.
Choice B reason: Discussing triggers extensively risks re-traumatization, intensifying flashbacks. Scientifically, premature focus on triggers heightens amygdala activity and cortisol, worsening PTSD symptoms. Stabilization through a calm environment is needed first to regulate emotional responses, ensuring readiness for trigger-focused therapy later in treatment.
Choice C reason: A calm environment and reassurance reduce hypervigilance and flashbacks by lowering amygdala activity. Scientifically, this stabilizes cortisol levels, promoting emotional regulation in PTSD. Creating safety mitigates trauma’s neurobiological impact, fostering trust and supporting initial recovery by preventing escalation of stress-related symptoms.
Choice D reason: Encouraging exposure to similar scenarios is premature, risking severe distress. Scientifically, uncontrolled exposure heightens amygdala-driven fear responses, exacerbating PTSD symptoms like flashbacks. Structured exposure therapy requires stabilization first, as premature desensitization can overwhelm coping mechanisms, delaying psychological recovery.
Correct Answer is ["A","B","C","D"]
Explanation
Choice A reason: Mental health resources and support groups address trauma’s psychological impact post-disaster. Scientifically, social support reduces cortisol and stabilizes amygdala-driven stress responses, fostering resilience. This mitigates acute stress or PTSD risk, supporting recovery by providing emotional and psychological resources for trauma processing.
Choice B reason: Risk assessment for self-harm identifies suicidal ideation, critical post-disaster. Trauma elevates cortisol and amygdala activity, increasing self-harm risk. Scientifically, thorough evaluation ensures safety, addressing neurobiological stress responses and guiding interventions to prevent harm, supporting psychological stability in crisis recovery.
Choice C reason: A therapeutic relationship fosters trust, encouraging disclosure post-disaster. Scientifically, trust reduces cortisol and stabilizes amygdala-driven fear, supporting emotional regulation. This facilitates engagement with interventions, addressing trauma’s psychological impact and promoting recovery by creating a safe environment for processing stress-related symptoms.
Choice D reason: Monitoring vital signs and consciousness tracks physiological stability post-disaster. Trauma can cause autonomic arousal, like elevated heart rate. Scientifically, regular monitoring detects stress-related changes, guiding interventions to stabilize neurobiological responses, ensuring safety and supporting recovery from trauma’s physical and psychological effects.
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