A patient with iron deficiency anemia has been prescribed ferrous sulfate. What is the expected pharmacologic action of ferrous sulfate?
Becomes part of hemoglobin and myoglobin to help carry and store oxygen throughout the body
Increases heart rate and blood pressure
Promotes calcium absorption in the bones
Enhances glucose metabolism
The Correct Answer is A
A. Becomes part of hemoglobin and myoglobin to help carry and store oxygen throughout the body: Ferrous sulfate provides elemental iron, which is incorporated into hemoglobin and myoglobin. This increases the oxygen-carrying capacity of red blood cells and improves tissue oxygenation, directly addressing iron deficiency anemia.
B. Increases heart rate and blood pressure: Ferrous sulfate does not have a direct effect on heart rate or blood pressure. Any changes in cardiovascular function are typically secondary to correction of anemia, not a pharmacologic action of the drug itself.
C. Promotes calcium absorption in the bones: Calcium absorption is not influenced by iron supplementation. Ferrous sulfate specifically targets hemoglobin and iron stores, not bone metabolism.
D. Enhances glucose metabolism: Iron supplementation does not affect glucose metabolism. Its therapeutic effect is limited to restoring iron levels and improving oxygen transport in the body.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. Slows down gastric emptying to concentrate glucose in the bloodstream:Glucagon does not act on the gastrointestinal tract to delay gastric emptying. Slowing gastric emptying is a mechanism associated with certain antidiabetic drugs like GLP-1 agonists, not glucagon.
B. Releases glycogen from the liver and muscles to be converted into glucose in the bloodstream:Glucagon stimulates glycogenolysis in the liver, breaking down stored glycogen into glucose, which is released into the bloodstream. This rapid increase in blood glucose makes it effective for treating severe hypoglycemia in unconscious patients.
C. Converts fats into triglycerides for ready conversion into glucose in the bloodstream:Glucagon does not convert fats into triglycerides. While glucagon can promote lipolysis, its primary pharmacologic action in hypoglycemia is glycogenolysis, not fat metabolism.
D. Releases glucose from the small intestine into the bloodstream:The small intestine absorbs dietary glucose but does not store it for rapid release. Glucagon’s effect is on liver glycogen stores, not intestinal glucose reserves.
Correct Answer is B
Explanation
A. Complete blood count with platelet count:Valproic acid can rarely cause thrombocytopenia, but yellowing of the eyes and skin (jaundice) indicates a potential liver issue rather than a blood cell abnormality. CBC is not the first priority here.
B. Liver function tests:Jaundice suggests hepatotoxicity, a known serious adverse effect of valproic acid. Liver function tests (AST, ALT, bilirubin) are essential to assess liver injury and guide whether the medication should be discontinued or adjusted.
C. Serum sodium level:Serum sodium is not related to the symptoms of jaundice. While valproic acid does not typically affect sodium levels, hyponatremia would not explain yellowing of the skin or eyes.
D. Blood glucose level:Blood glucose monitoring is unrelated to valproic acid-induced jaundice. Hyperglycemia or hypoglycemia would not present with yellowing of the skin or eyes.
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