Contractility refers to the
The Correct Answer is {"dropdown-group-1":"D"}
A. Rate: The rate of contraction refers to the heart rate, which is determined by the frequency of action potentials generated by the sinoatrial node. While rate influences cardiac output, it does not describe the intrinsic strength of myocardial contraction.
B. Frequency: Frequency also relates to how often the heart beats over a period of time, essentially the number of cardiac cycles per minute. It does not reflect the force generated by the myocardial fibers during systole.
C. Speed: Speed could be interpreted as the velocity of contraction or conduction, but it does not measure the intrinsic strength of the ventricular contraction. Contractility is independent of how quickly the contraction occurs.
D. Force: Contractility refers to the inherent ability of cardiac muscle fibers to generate force during systole, independent of preload and afterload. It reflects the efficiency of actin-myosin cross-bridge formation and calcium handling within the myocardium, determining how strongly the heart can pump blood with each beat.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is {"dropdown-group-1":"C","dropdown-group-2":"A"}
Explanation
Correct answer:
- Stroke volume (SV) = 60 mL
- This stroke volume is within the normal expected range.
Stroke volume represents the amount of blood ejected from the ventricle with each heartbeat and is calculated using the formula:
SV = EDV – ESV
EDV (end-diastolic volume) = 150 mL
ESV (end-systolic volume) = 90 mL
SV = 150 mL – 90 mL
= 60 mL
An anatomically and physiologically normal adult stroke volume ranges from approximately 60–100 mL per beat. This measurement indicates that the ventricles are ejecting an adequate volume of blood during systole. The calculation reflects effective ventricular filling and contractility, suggesting that the patient’s heart is pumping a normal volume of blood per beat.
Correct Answer is A
Explanation
Correct answer: True
The Bainbridge reflex is a physiological response mediated by stretch receptors located in the walls of the right atrium and the venae cavae. When venous return increases, atrial stretch stimulates these proprioceptive receptors, sending signals via the vagus nerve to the cardiovascular centers in the medulla oblongata. In response, sympathetic activity to the sinoatrial node increases, resulting in an elevated heart rate. This reflex helps maintain homeostasis by ensuring that the increased blood volume returning to the heart is effectively ejected, preventing venous congestion and optimizing cardiac output.
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