During the first stage of increased intracranial pressure, the brain attempts to compensate by employing a mechanism called:
Rejuvenation
Autoregulation
Percolation
Demarcation
The Correct Answer is B
A. Rejuvenation –. This is not a physiological term used in the context of ICP or brain compensation.
B. Autoregulation –This is the brain’s ability to maintain consistent cerebral blood flow (CBF) despite changes in systemic blood pressure. In early stages of increased ICP, autoregulation helps maintain oxygen and nutrient delivery to brain tissue by adjusting cerebral vessel diameter.
C. Percolation –This term is not relevant in neurophysiology or ICP management.
D. Demarcation –This refers more to distinguishing boundaries between tissues (e.g., in gangrene), not compensatory mechanisms for ICP.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
A. Ventricular fibrillation is a life-threatening arrhythmia, not a compensatory mechanism for heart failure.
B. Increased intracranial pressure (ICP) is unrelated to heart failure compensation.
C. The renin-angiotensin-aldosterone system (RAAS) is activated in heart failure to maintain blood pressure and perfusion. This leads to sodium and water retention, causing fluid buildup and edema.
D. Atrial flutter is a cardiac arrhythmia, not a compensatory mechanism causing fluid retention.
Correct Answer is C
Explanation
A. A thin and strong left ventricle is not typical of systolic heart failure.
B. A thick and weak ventricle is more characteristic of diastolic heart failure (impaired relaxation and filling).
C. Systolic heart failure involves impaired contraction of the left ventricle, which often becomes thin and weak due to ventricular dilation and loss of contractile strength.
D. A thick and strong ventricle is not typical of heart failure.
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