During the first stage of increased intracranial pressure, the brain attempts to compensate by employing a mechanism called:
Rejuvenation
Autoregulation
Percolation
Demarcation
The Correct Answer is B
A. Rejuvenation –. This is not a physiological term used in the context of ICP or brain compensation.
B. Autoregulation –This is the brain’s ability to maintain consistent cerebral blood flow (CBF) despite changes in systemic blood pressure. In early stages of increased ICP, autoregulation helps maintain oxygen and nutrient delivery to brain tissue by adjusting cerebral vessel diameter.
C. Percolation –This term is not relevant in neurophysiology or ICP management.
D. Demarcation –This refers more to distinguishing boundaries between tissues (e.g., in gangrene), not compensatory mechanisms for ICP.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. Blood urea nitrogen assesses kidney function but is less directly related to digoxin toxicity risk.
B. Creatinine also reflects kidney function but is not the highest priority in this context.
C. Hemoglobin levels are not directly related to digoxin administration.
D. Potassium levels are critical to review because hypokalemia increases the risk of digoxin toxicity, and hyperkalemia can also affect the drug's efficacy. Monitoring potassium is essential before giving digoxin.
Correct Answer is D
Explanation
A. Furosemide typically causes hypokalemia (low potassium), not hyperkalemia.
B. There is no such thing as a potassium allergy; this statement is incorrect.
C. Furosemide does not increase potassium levels; it promotes potassium excretion.
D. Furosemide is a loop diuretic that increases the excretion of potassium in urine, so patients often require potassium supplementation to prevent hypokalemia.
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