The nurse is caring for a client with septic shock and assesses blood oozing out from the peripheral intravenous site, bleeding gums, and blood in the indwelling urinary catheter bag. Which intervention would the nurse implement first?
Check the client’s hemoglobin & hematocrit level
Obtain a STAT electrocardiogram (EKG)
Monitor the client’s temperature
Check the client’s platelets, PT, and INR
The Correct Answer is D
Choice A reason: Hemoglobin and hematocrit levels are indicators of the total red blood cell mass and oxygen carrying capacity. While these values are important to assess the severity of blood loss already sustained, they do not help identify the underlying coagulopathy causing the spontaneous bleeding from multiple unrelated sites.
Choice B reason: A STAT electrocardiogram is used to assess cardiac rhythm and identify myocardial ischemia or conduction abnormalities. While septic shock can place significant strain on the cardiovascular system, an EKG is not the priority diagnostic tool for a patient exhibiting signs of systemic, multi-site spontaneous hemorrhage.
Choice C reason: Monitoring the client’s temperature is a standard part of assessing a patient with sepsis to track the inflammatory response or the effectiveness of antibiotic therapy. However, thermoregulation assessment does not address the immediate life threatening risk associated with suspected disseminated intravascular coagulation and active bleeding.
Choice D reason: The clinical presentation of oozing from IV sites, bleeding gums, and hematuria in a septic patient is highly suggestive of disseminated intravascular coagulation. Checking the platelet count, Prothrombin Time, and International Normalized Ratio is the critical first step to confirm a consumption coagulopathy and guide blood product replacement.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: Septic shock is defined by persistent hypotension that does not respond to intravenous fluid resuscitation, requiring vasopressors to maintain a mean arterial pressure ≥ 65 mm Hg. A drop in blood pressure to 70/34 despite a bolus is a hallmark indicator of the transition to shock.
Choice B reason: A decrease in white blood cell count toward the normal range (5,000 to 10,000/mm3) generally suggests an improving inflammatory or infectious state. In severe sepsis, a very low WBC (leukopenia) might be seen, but a shift from 11,000 to 9,500 is not a primary indicator of shock.
Choice C reason: While an increase in lactate suggests worsening tissue perfusion, a value of 1.8 mmol/L is still within the normal range (typically < 2.0 mmol/L). In septic shock, one would expect the lactate to rise significantly, usually above 2.0 mmol/L, as anaerobic metabolism becomes more widespread.
Choice D reason: A urine output of 300 mL following a fluid bolus is an excellent clinical sign, suggesting that the kidneys are being well perfused and are responding to the volume expansion. This finding would indicate an improvement in hemodynamic status rather than a progression toward shock.
Correct Answer is A
Explanation
Choice A reason: Cardiogenic shock occurs when the heart’s muscular wall fails to pump effectively. A myocardial infarction (MI) causes necrosis of the cardiac tissue; if more than 40% of the left ventricle is damaged, the heart cannot maintain adequate cardiac output, leading directly to cardiogenic shock.
Choice B reason: A client with trauma-related blood loss is at risk for hypovolemic shock. This type of shock is caused by a lack of intravascular volume rather than a primary failure of the heart muscle itself, distinguishing it from the pump-failure mechanism of cardiogenic shock.
Choice C reason: A tension pneumothorax causes obstructive shock. The high pressure in the thoracic cavity shifts the mediastinum and compresses the great vessels (superior and inferior vena cava), physically preventing blood from returning to the heart, which is different from a primary muscular failure.
Choice D reason: A client with positive blood cultures is at risk for septic shock. This is a form of distributive shock caused by systemic vasodilation and increased capillary permeability in response to an overwhelming infection, rather than an initial failure of the cardiac pump.
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