The nurse is caring for a patient on mechanical ventilation. Which of the following must the nurse implement to prevent ventilator-associated pneumonia (VAP)? Drag and Drop Directions: Click and drag the correct answer choices from the Possible Answers box on the left to the Correct Answers box on the right. More than one answer may be chosen.
Provide oral care every 12 hours
Monitor ETT cuff pressure
Insert a nasogastric tube
Administer high doses of sedation
Maintain head of the bed to at least 30 degrees
Correct Answer : B,E
A. Provide oral care every 12 hours: Oral care is an essential component of VAP prevention; however, every 12 hours is insufficient for mechanically ventilated patients. Evidence-based bundles recommend more frequent oral care, often every 2–4 hours, typically with chlorhexidine to reduce oropharyngeal colonization. This frequency does not meet best-practice standards.
B. Monitor ETT cuff pressure: Maintaining appropriate endotracheal tube cuff pressure (usually 20–30 cm H₂O) helps prevent microaspiration of contaminated oral and gastric secretions into the lower airway. Inadequate cuff pressure increases the risk of pathogen entry into the lungs, making regular monitoring a critical VAP prevention strategy.
C. Insert a nasogastric tube: Insertion of a nasogastric tube does not prevent VAP and may increase the risk of aspiration if not managed properly. Gastric distention and reflux can contribute to aspiration of gastric contents, thereby increasing pneumonia risk rather than reducing it.
D. Administer high doses of sedation: High sedation levels suppress cough reflexes, impair early mobilization, and prolong mechanical ventilation duration, all of which increase VAP risk. Current evidence supports daily sedation interruption and light sedation strategies rather than deep sedation.
E. Maintain head of the bed to at least 30 degrees: Elevating the head of the bed between 30–45 degrees reduces the risk of aspiration of gastric and oral secretions. This positioning decreases the likelihood of bacteria entering the lower respiratory tract and is a cornerstone of ventilator bundle protocols.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. Echocardiogram results with an ejection fraction of 55%: An ejection fraction (EF) of 55% is within the normal range, indicating preserved systolic function. While some forms of heart failure, such as HFpEF (heart failure with preserved ejection fraction), can occur, a normal EF alone does not strongly suggest acute heart failure as the cause of shortness of breath.
B. BNP results of 155 pg/mL (reference range <100 pg/mL): Brain natriuretic peptide (BNP) is released in response to ventricular stretch and increased intracardiac pressures. A BNP of 155 pg/mL is elevated, supporting a cardiac etiology of dyspnea, such as acute or chronic heart failure. BNP is a sensitive and specific biomarker for distinguishing heart failure from other causes of shortness of breath.
C. EKG results of sinus rhythm with occasional premature ventricular contractions (PVCs): Sinus rhythm with occasional PVCs does not indicate heart failure. While arrhythmias may coexist with heart failure, this finding alone does not explain the patient’s dyspnea or support a heart failure diagnosis.
D. ABG result with a PaCO2 of 30 mmHg (reference range 35-45 mmHg): A low PaCO2 reflects hyperventilation, which may be a compensatory response to hypoxia or pulmonary pathology. While it provides information on respiratory status, it is nonspecific and does not directly indicate heart failure as the underlying cause of dyspnea.
Correct Answer is ["A","B","D","E"]
Explanation
A. Tachycardia: An elevated heart rate is a compensatory response to hypoxemia or increased work of breathing. In COPD exacerbations, tachycardia often accompanies early respiratory distress and signals the body’s attempt to maintain oxygen delivery to tissues.
B. Dyspnea: Shortness of breath or difficulty breathing is a primary and direct indicator of respiratory compromise. Progressive dyspnea reflects increased airway resistance, impaired gas exchange, and impending respiratory failure if not addressed promptly.
C. Muscle Tremors: Muscle tremors are not typically a direct sign of respiratory distress. They may occur from anxiety, medications such as beta-agonists, or metabolic disturbances, but they are not a reliable indicator of worsening pulmonary function.
D. Diaphoretic: Excessive sweating is a common autonomic response to hypoxia, increased work of breathing, or sympathetic activation. Diaphoresis in the context of COPD exacerbation signals significant physiologic stress and potential respiratory compromise.
E. Unable to speak in full sentences: Inability to complete sentences without pausing to breathe indicates severe respiratory effort and decreased ventilatory reserve. This is a critical sign of impending respiratory failure requiring immediate assessment and intervention.
F. Nausea: Nausea may occur due to medications, hypoxia, or anxiety but is not a primary manifestation of respiratory distress. It is a nonspecific symptom and less relevant in assessing acute respiratory compromise.
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