The nurse teaches a client with chronic pancreatitis how to monitor the effectiveness of pancreatic enzyme replacement therapy. Which instruction should the nurse include?
"Record your daily fluid intake.
"Observe your stools for steatorrhea."
"Test your urine for ketones once a day."
"Perform glucose fingerstick tests twice a day."
The Correct Answer is B
Rationale:
A. Tracking fluid intake is generally part of maintaining overall hydration and managing general health, but it does not provide information about how well pancreatic enzymes are working. Pancreatic enzyme replacement therapy (PERT) is aimed at improving digestion, particularly fat absorption, rather than influencing fluid balance. Therefore, while hydration is important, it is not an indicator of therapy effectiveness.
B. The main purpose of PERT is to replace digestive enzymes that the pancreas is no longer producing adequately, particularly lipase, amylase, and protease, to aid in digestion of fats, carbohydrates, and proteins. Steatorrhea—characterized by bulky, oily, pale, or foul-smelling stools—is a direct sign of fat malabsorption. By observing changes in stool consistency, color, and frequency, the client can assess whether the enzyme therapy is effectively aiding digestion. A decrease in steatorrhea indicates the therapy is working, while persistent fatty stools suggest the need for dosage adjustment or adherence review.
C. Urine ketone testing is used primarily for clients at risk for diabetic ketoacidosis or during prolonged fasting/starvation. It reflects fat breakdown for energy due to insufficient insulin, not fat malabsorption. Therefore, it is unrelated to assessing the effectiveness of pancreatic enzymes in chronic pancreatitis.
D. Clients with chronic pancreatitis may develop diabetes mellitus due to pancreatic damage, making glucose monitoring important for glycemic control. However, glucose levels do not indicate whether pancreatic enzyme therapy is effectively digesting fats, proteins, and carbohydrates. Monitoring stool characteristics remains the most direct and clinically relevant measure.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Rationale:
A. Lactulose can cause loose stools, but diarrhea is not the desired therapeutic outcome. Excessive diarrhea can lead to dehydration, electrolyte imbalances, and worsening confusion, which are adverse effects rather than expected responses.
B. Lactulose is used to reduce serum ammonia levels in clients with hepatic encephalopathy by promoting excretion of ammonia through the stool. The expected therapeutic outcome is regular, soft bowel movements, typically two to five per day, without causing excessive diarrhea. This indicates that ammonia is being cleared effectively, reducing neurotoxic effects.
C. Improvement in mental status is the ultimate goal of lactulose therapy, but it may take several doses to achieve. Immediate changes in speech are not expected and do not solely indicate treatment effectiveness.
D. Fetor hepaticus (a characteristic musty breath odor) and asterixis (flapping tremor of the hands) are signs of worsening hepatic encephalopathy. Their appearance would indicate treatment failure or progression of disease, not a positive response.
Correct Answer is D
Explanation
Rationale:
A. Heat exhaustion typically presents with normal to slightly elevated body temperature, usually below 40°C. A core temperature above 40°C indicates heat stroke, a more severe heat-related illness that involves central nervous system dysfunction and potential organ failure. Therefore, a temperature of 40.9°C is not expected in heat exhaustion.
B. While tachycardia and hypotension can occur in heat exhaustion due to fluid and electrolyte loss, hot, dry skin is a hallmark of heat stroke, not heat exhaustion. In heat exhaustion, the body continues to sweat profusely as a compensatory mechanism to dissipate heat. The presence of dry skin would suggest progression to a more severe condition.
C. Profuse sweating and tachycardia are indeed common in heat exhaustion, reflecting dehydration and sympathetic nervous system activation. However, confusion or altered mental status is more characteristic of heat stroke, indicating central nervous system involvement. In true heat exhaustion, cognitive function is generally preserved, though the client may feel weak or fatigued.
D. These are classic signs of heat exhaustion, resulting from fluid and electrolyte loss and decreased perfusion due to prolonged heat exposure. Additional symptoms may include fatigue, muscle cramps, mild tachycardia, and pallor. The presence of intact mental status differentiates heat exhaustion from heat stroke. Early recognition and management with oral or IV fluids, cooling measures, and rest in a cool environment are essential to prevent progression.
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