A nurse is educating a group of adolescents about the causes and risk factors associated with Acne Vulgaris. Which statement accurately explains a risk factor for this condition?
"Excessive exposure to sunlight can lead to Acne Vulgaris.".
"Genetic predisposition is a risk factor for Acne Vulgaris.".
"Consuming a diet high in vitamin C can cause Acne Vulgaris.".
"Wearing sunscreen regularly can increase the risk of Acne Vulgaris.".
The Correct Answer is B
Choice A rationale:
Excessive exposure to sunlight does not lead to Acne Vulgaris.
In fact, sunlight exposure can have a drying effect on the skin and may temporarily improve acne symptoms in some individuals.
However, it is not a direct cause of the condition.
Choice B rationale:
Genetic predisposition is a well-established risk factor for Acne Vulgaris.
If a person has a family history of acne, they are more likely to develop the condition themselves.
Genetic factors can influence the individual's skin type, oil production, and susceptibility to inflammation, all of which contribute to the development of acne.
Choice C rationale:
Consuming a diet high in vitamin C does not cause Acne Vulgaris.
In fact, vitamin C is known for its antioxidant properties and is generally considered beneficial for skin health.
Acne is primarily driven by factors such as genetics, hormones, and skin bacteria, not vitamin C intake.
Choice D rationale:
Wearing sunscreen regularly does not increase the risk of Acne Vulgaris.
Sunscreen is essential for protecting the skin from harmful UV radiation and preventing conditions like sunburn and skin cancer.
It does not play a role in the development of acne.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A rationale:
Itchy, scaly patches of skin are not commonly associated with Acne Vulgaris.
Instead, these symptoms may be indicative of other skin conditions such as eczema or psoriasis.
Choice B rationale:
The formation of comedones (blackheads and whiteheads) is a hallmark symptom of Acne Vulgaris.
These comedones result from the clogging of hair follicles with a combination of sebum and dead skin cells.
Choice C rationale:
Skin redness and blistering are not typical symptoms of Acne Vulgaris.
These symptoms may suggest other skin conditions or reactions to topical products but are not characteristic of acne.
Choice D rationale:
Hair loss and thinning are not commonly associated with Acne Vulgaris.
Hair loss is more often related to conditions such as alopecia or hormonal imbalances, rather than acne.
Correct Answer is B
Explanation
Choice A rationale:
The presence of redness, swelling, and tenderness.
Rationale: Closed comedones, also known as whiteheads, are non-inflammatory lesions that do not exhibit redness, swelling, or tenderness.
These lesions are characterized by the accumulation of sebum and dead skin cells within hair follicles, which form a white or flesh-colored plug that is not associated with inflammation.
Choice B rationale:
Closed comedones (whiteheads).
Rationale: Closed comedones, or whiteheads, are commonly observed in non-inflammatory presentations of acne vulgaris.
These lesions occur when hair follicles become blocked with sebum and dead skin cells, leading to the formation of a white or flesh-colored plug.
Unlike inflammatory lesions, whiteheads do not exhibit redness, swelling, or tenderness.
Choice C rationale:
Nodules and cysts.
Rationale: Nodules and cysts are typically observed in severe cases of acne vulgaris and represent inflammatory lesions.
These lesions are characterized by deep, painful, and often pus-filled nodules or cysts beneath the skin's surface.
They are not commonly associated with non-inflammatory presentations.
Choice D rationale:
Papules and pustules.
Rationale: Papules and pustules are also inflammatory lesions seen in acne vulgaris.
Papules are small, raised, red bumps, while pustules are similar but contain pus.
These lesions are associated with redness, swelling, and tenderness, which are not characteristic of non-inflammatory lesions.
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