A nurse is providing vehicle safety education to the parents of a premature newborn.
Which of the following statements should the nurse include in the teaching?

Choice A rationale: The client exhibits signs consistent with severe preeclampsia including blood pressure above 160/110 mm Hg, 3+ to 4+ deep tendon reflexes, and positive clonus, which are neurological indicators of central nervous system irritability. These findings increase the risk for eclamptic seizures. The headache, visual disturbances (“spots”), and epigastric pain further indicate imminent seizure risk from cerebral edema and vasospasm. Early intervention is crucial to prevent progression to eclampsia.

Choice B rationale: Elevated blood pressure combined with preeclampsia significantly raises the risk of abruptio placentae, the premature separation of the placenta from the uterine wall. This occurs because hypertension causes vasospasm and weakening of the placental attachment, increasing placental insufficiency and hemorrhage. The client’s edema and headache indicate vascular endothelial dysfunction, contributing to this risk. Abruptio placentae is a critical obstetric emergency requiring urgent recognition.

Choice C rationale: The client’s symptoms and signs of hypertension, headache, epigastric pain, and elevated blood pressure with edema suggest progression toward HELLP syndrome (Hemolysis, Elevated Liver enzymes, Low Platelets). Although labs are not provided, the clinical presentation fits the syndrome’s prodromal phase. HELLP syndrome is a severe variant of preeclampsia with hepatic involvement, which can cause multiorgan dysfunction and increased maternal-fetal morbidity if untreated.

Choice D rationale: Polyuria, or excessive urine output, is not typical in preeclampsia or related hypertensive disorders; instead, oliguria (decreased urine output below 30 mL/hr) may occur due to renal impairment. The client’s urine output of 25–55 mL/hr borders low normal, signaling possible kidney hypoperfusion, but polyuria is not a recognized complication here. Polyuria is more associated with diabetes or diuretic therapy, not hypertensive pregnancy disorders.

Choice E rationale: Ketoacidosis is metabolic acidosis due to ketone accumulation from uncontrolled diabetes or starvation. This client has no history or symptoms of diabetes or starvation, and no laboratory evidence of hyperglycemia or acidosis is reported. Ketoacidosis is not a complication linked to preeclampsia or hypertension in pregnancy and is therefore unlikely in this case.

Choice F rationale: Fetal macrosomia is excessive fetal growth usually related to maternal diabetes or obesity. This client has no indications of gestational diabetes or excessive fetal growth; fetal heart rate and contractions are within normal limits, and no mention of abnormal fundal height or ultrasound findings is given. The mild hypertension and edema do not predispose to macrosomia.

Abruptio Placentae: Typically does not cause increased contraction frequency; contractions may be normal or decreased due to uterine irritation or pain.

Uterine Tachysystole: Characterized by more than 5 contractions in 10 minutes averaged over 30 minutes, indicating excessive contraction frequency, which can reduce uteroplacental perfusion.

Uterine Tone
Abruptio Placentae: The uterus often becomes firm, rigid, and tender due to bleeding and inflammation caused by premature placental separation, increasing baseline uterine tone.

Uterine Tachysystole: Uterine tone is elevated because contractions are prolonged and close together, causing the uterus to remain tense with insufficient relaxation.

Pain Report
Abruptio Placentae: Patients usually report sharp, severe abdominal or back pain due to the sudden placental detachment and uterine irritation.

Uterine Tachysystole: Pain may be present but is typically related to frequent contractions rather than sharp, localized pain.

FHR Pattern
Abruptio Placentae: Commonly causes fetal heart rate abnormalities like late decelerations, bradycardia, or absent variability due to fetal hypoxia from compromised placental perfusion.

Uterine Tachysystole: Can cause decreased fetal heart rate variability and late decelerations because frequent contractions reduce oxygen delivery during inadequate relaxation.