A patient is diagnosed with the type of diabetes in which the plasma beta cells fail to respond to insulin. Which type of diabetes is this patient experiencing?
Gestational diabetes
Type 1 diabetes mellitus
Type 2 diabetes mellitus
Impaired glucose tolerance
The Correct Answer is C
Choice A reason: Gestational diabetes involves insulin resistance due to placental hormones, not primarily beta cell failure to respond to insulin. It occurs during pregnancy and usually resolves post-delivery, making it distinct from type 2 diabetes, where insulin resistance and beta cell dysfunction are chronic.
Choice B reason: Type 1 diabetes results from autoimmune destruction of pancreatic beta cells, leading to absolute insulin deficiency, not beta cells failing to respond to insulin. The lack of insulin production distinguishes it from type 2, where insulin resistance is the primary issue.
Choice C reason: Type 2 diabetes is characterized by insulin resistance, where peripheral tissues and beta cells fail to respond effectively to insulin, combined with progressive beta cell dysfunction. This leads to relative insulin deficiency, making this the correct diagnosis for the described pathophysiology.
Choice D reason: Impaired glucose tolerance is a prediabetic state with elevated glucose levels but not yet meeting diabetes criteria. It involves mild insulin resistance but not significant beta cell failure to respond to insulin, distinguishing it from established type 2 diabetes with more pronounced dysfunction.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: A blood glucose of 750 mg/dL indicates severe hyperglycemia, potentially diabetic ketoacidosis (DKA) or hyperosmolar hyperglycemic state (HHS), both life-threatening. Notifying the RN and provider ensures rapid intervention, such as IV insulin and fluids, to correct glucose, acidosis, and dehydration, preventing multi-organ failure or coma.
Choice B reason: Verifying diabetes type is important but not the priority with a glucose of 750 mg/dL. Both type 1 (DKA risk) and type 2 (HHS risk) require urgent treatment for severe hyperglycemia. Delaying action for record review risks worsening metabolic decompensation, making this less urgent.
Choice C reason: Administering sliding scale insulin without provider guidance is unsafe for severe hyperglycemia (750 mg/dL), as it may be insufficient for DKA or HHS. IV insulin and fluid therapy are typically required, and acting independently risks inadequate treatment, delaying critical care and worsening patient outcomes.
Choice D reason: Assessing vital signs is useful but not the priority. Severe hyperglycemia requires immediate medical intervention to correct metabolic derangements. Vital signs may indicate shock or dehydration, but notifying the provider ensures comprehensive management, including diagnostics and treatment, addressing the root cause more effectively.
Correct Answer is C
Explanation
Choice A reason: Hyperglycemia typically increases appetite (polyphagia) due to cellular starvation, as glucose cannot enter cells effectively without insulin. Decreased appetite is not a hallmark of hyperglycemia and may indicate other issues, like gastrointestinal complications, making this statement incorrect for understanding hyperglycemia manifestations.
Choice B reason: Blurred vision can occur in hyperglycemia due to osmotic swelling of the lens from high glucose levels, but it is not constant ("all of the time"). This statement exaggerates the symptom, indicating a partial misunderstanding, as vision changes are typically temporary and resolve with glucose control.
Choice C reason: Increased thirst (polydipsia) is a classic hyperglycemia symptom. High blood glucose causes osmotic diuresis, leading to dehydration, which stimulates thirst to compensate for fluid loss. This statement reflects accurate understanding of hyperglycemia’s physiological effects on fluid balance and renal function.
Choice D reason: Fruity breath odor is a sign of diabetic ketoacidosis, a complication of severe hyperglycemia, due to ketone production from fat metabolism. This indicates understanding of a critical hyperglycemia manifestation, as it reflects metabolic acidosis requiring urgent intervention, making this statement correct.
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