The LPN is caring for a client that was recently started on furosemide for dependent edema caused by right sided heart failure. Which nursing actions are appropriate to add to the plan of care for the patient?

A. Myocardial infarction: While this patient has significant cardiovascular risk factors, the clinical presentation does not describe acute substernal chest pain or EKG changes. The presence of 4+ peripheral edema and jugular venous distention points toward a chronic congestive process rather than acute coronary occlusion. Weight gain and exhaustion are symptoms of volume overload.

B. Left-sided heart failure: Left-sided failure typically presents with pulmonary congestion, characterized by crackles, orthopnea, and paroxysmal nocturnal dyspnea. While it often precedes right-sided failure, the specific findings of jugular distention and lower extremity edema are hallmarks of systemic venous congestion. This choice does not align with the predominant systemic symptoms described.

C. Acute pericarditis: Pericarditis usually presents with sharp, pleuritic chest pain that improves when leaning forward, often accompanied by a pericardial friction rub. It does not typically cause massive peripheral edema or rapid weight gain unless it progresses to cardiac tamponade. The clinical signs in the stem indicate high systemic venous pressure.

D. Right-sided heart failure: This condition results in the inability of the right ventricle to pump blood into the pulmonary circulation, causing systemic backup. Jugular venous distention, 4+ pitting edema, and rapid weight gain are classic signs of systemic venous hypertension. The exhaustion stems from decreased cardiac output during physical exertion.

A. Diltiazem: This calcium channel blocker reduces blood pressure and slows the heart rate, both of which can contribute to orthostatic hypotension. In a 70-year-old patient, the baroreceptor reflex is less efficient, making them more susceptible to syncopal episodes during position changes. Lowering systemic vascular resistance too much can result in an acute drop in cerebral perfusion.

B. Carbidopa/levodopa: This medication is primarily used to treat Parkinson's disease and, while it can cause some hypotension, it is not a primary cardiovascular agent. Its impact on blood pressure is generally less profound than that of diuretics or dedicated antihypertensives. It is less likely than the other choices to be the primary driver of a blood pressure of 88/48.

C. Hydrochlorothiazide: Diuretics reduce the total intravascular volume by promoting the excretion of sodium and water. A decrease in blood volume directly lowers the blood pressure and can lead to dehydration-related syncope. This medication is a very common cause of orthostatic dizziness and falls in the geriatric population.

D. Metoprolol: As a beta-blocker, metoprolol reduces cardiac output and inhibits the compensatory tachycardia usually seen when standing. This prevents the body from quickly adjusting the blood pressure during postural changes, leading to the reported dizziness. The combination of reduced heart rate and reduced force of contraction contributes to the hypotensive state.

E. aspirin: Aspirin is an antiplatelet agent used for the prevention of cardiovascular events and does not have a direct effect on blood pressure. It does not cause vasodilation, volume depletion, or changes in heart rate that would lead to syncope. Its primary side effects are related to gastrointestinal irritation and bleeding risks rather than hemodynamics.