What is the nurse’s best response when a client with anemia asks “Why am I feeling tired all the time?
“Your tissues are not getting enough oxygen?”
"When you are sick, you need to rest more.”
"How many hours are you sleeping at night?"
“You are probably dehydrated.”
The Correct Answer is A
Anemia is characterized by a deficit in hemoglobin, the primary protein in erythrocytes responsible for binding and transporting oxygen. When the concentration of hemoglobin falls below physiological requirements (typically < 12-13 g/dL), the blood's oxygen-carrying capacity is significantly diminished. This results in inadequate delivery of oxygen to meet the metabolic demands of muscles and organs, leading to persistent, systemic exhaustion.
Rationale:
A. The nurse’s best response is, “Your tissues are not getting enough oxygen.” This accurately explains the pathophysiology of anemia in simple, scientific terms. Without sufficient hemoglobin, the blood cannot transport the oxygen needed for cellular respiration and energy production. This lack of fuel at the cellular level is the direct cause of the profound fatigue the client experiences.
B. Telling the client they need to rest more because they are sick is a dismissive and non-scientific response. While rest is helpful, it does not explain the biological cause of the tiredness. A client with anemia is not simply "tired" from activity; they are suffering from a functional deficit in their blood's ability to support metabolic activity.
C. Asking about sleep hours is a general assessment question for insomnia but does not address the client's question about their anemia. Anemic fatigue is distinct from sleepiness; no amount of sleep can correct the underlying problem of inadequate oxygenation. The nurse should focus on the disease process rather than the client's sleep habits in this context.
D. Attributing the fatigue to dehydration is medically incorrect. While dehydration can cause lethargy, it is not the cause of fatigue in a patient specifically diagnosed with anemia. Providing misinformation can confuse the client about their treatment plan. The focus must remain on the hemoglobin deficiency and its impact on the body's aerobic capacity.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Acute pancreatitiscan lead to necrotizing changes that erode major abdominal blood vessels, resulting in retroperitoneal hemorrhage. This leads to a rapid loss of intravascular volume, manifesting as hypovolemic shock. Early recognition of shifting hemodynamic parameters is critical, as hemorrhagic pancreatitis has a high mortality rate due to the combination of blood loss and systemic inflammatory response syndrome.
Rationale:
A.Electrolyte imbalances, specifically hypocalcemia, are common in pancreatitis due to fat necrosis and soap formation. However, they do not typically manifest with this specific triad of hypotension, tachycardia, and tachypnea. These vital signs indicate a primary circulatory failure rather than a purely chemical or mineral imbalance in the blood.
B.Pleural effusion can occur in pancreatitis as inflammatory exudate travels through the diaphragm, causing respiratory distress and tachypnea. While the patient's respirations are elevated (28/min), pleural effusion alone does not explain the significant hypotension (92/50). The combination of low blood pressure and high heart rate suggests a volume-related vascular emergency.
C.The nurse should suspect internal bleeding(hemorrhagic pancreatitis). The combination of tachycardia (116 bpm) and hypotension (92/50) are classic indicators of hypovolemic shock. Pancreatic enzymes can digest the walls of adjacent blood vessels, leading to massive retroperitoneal hemorrhage. This is a critical complication requiring aggressive fluid resuscitation and potentially surgical or radiological intervention.
D.A pancreatic pseudocyst is a localized collection of fluid, enzymes, and debris that forms over weeks. While it can cause pain or become infected (abscess), it does not cause an acute hemodynamic collapse. The vital signs provided in the question indicate an acute, life-threatening vascular event rather than a subacute or chronic fluid collection complication.
Correct Answer is A
Explanation
Maintaining fluid homeostasisis a critical challenge in chronic kidney disease due to the progressive loss of nephron function and reduced glomerular filtration rate. When the kidneys can no longer excrete excess water, fluid shifts into the interstitial spaces and pulmonary vasculature. A stable fluid balance indicates that the patient's restricted intake is matched by their limited output, preventing life-threatening pulmonary congestion.
Rationale:
A.The absence of adventitious soundsin the lungs, such as crackles or rales, is a primary indicator that the client’s fluid balance is stable. This suggests that fluid is not accumulating in the pulmonary interstitial spaces or alveoli. It confirms that the current fluid restriction is effective in preventing extracellular fluid volume excessand potential respiratory failure.
B.Decreased calcium levels (hypocalcemia) are a common finding in CKD due to impaired vitamin D activation and hyperphosphatemia, but they do not indicate fluid stability. Electrolyte imbalances in renal disease are chemical markers of metabolic bone disease rather than direct indicators of volume status. A patient can have stable fluid levels while still possessing severe mineral imbalances.
C.Increased edema in the legs is a clear sign of fluid volume excess and instability. This indicates that the current fluid restriction is insufficient or that the patient is not adhering to the regimen. Edema reflects the migration of fluid into the interstitial spacedue to increased hydrostatic pressure, signifying that the patient's fluid balance is worsening.
D.Increased phosphorus levels (hyperphosphatemia) occur in CKD because the kidneys cannot excrete phosphate effectively. Like calcium, phosphorus is a marker of mineral and bone disorder and renal clearance efficiency. While it is an important laboratory finding to monitor in CKD, it provides no information regarding the stability of the patient's actual fluid volume status.
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