Which of the following are priorities in the treatment and nursing management of the patient with an acute myocardial infarction (AMI)?
Keep oxygen saturation levels below 89
Diurese aggressively and monitor daily weight.
Provide adequate pain control to reduce myocardial oxygen demands.
Administer thrombolytic therapy if indicated.
Correct Answer : C,D
Choice A rationale
Keeping oxygen saturation levels below 89 percent is physiologically detrimental and scientifically incorrect for a patient with an acute myocardial infarction. The goal of therapy is to maintain SpO2 ≥ 94 percent to ensure the myocardium receives sufficient oxygen to prevent further cell death. Hypoxia triggers a sympathetic nervous system response, increasing heart rate and contractility, which elevates myocardial oxygen demand. Chronic low saturation leads to anaerobic metabolism, lactic acid production, and eventual cardiogenic shock.
Choice B rationale
Aggressive diuresis is not a universal priority for all patients with acute myocardial infarction unless they are showing clinical signs of pulmonary edema or volume overload. While monitoring daily weight is a standard nursing practice for heart failure, the primary goal in the acute phase is restoring perfusion rather than massive fluid removal. Over-diuresis can lead to hypovolemia and decreased cardiac output, which compromises coronary artery perfusion and can worsen the extent of the myocardial injury.
Choice C rationale
Pain control is a critical priority because chest pain triggers the release of catecholamines like epinephrine and norepinephrine. These hormones increase heart rate, blood pressure, and myocardial contractility, all of which substantially raise the amount of oxygen the heart requires. By administering morphine or nitroglycerin to alleviate pain, the nurse effectively lowers the metabolic demand of the damaged heart muscle. This balance between oxygen supply and demand is essential for limiting the final size of the infarct.
Choice D rationale
Thrombolytic therapy is a primary medical intervention aimed at restoring coronary blood flow by dissolving the fibrin clot that caused the myocardial infarction. Scientific evidence supports the use of these agents within a specific time window to reperfuse ischemic tissue and salvage myocardium. Restoring patency to the occluded vessel prevents the progression of ischemia to irreversible necrosis. This intervention is specifically prioritized in ST-elevation myocardial infarction cases where primary percutaneous coronary intervention is not immediately available.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A rationale
Sinus tachycardia is characterized by a normal cardiac conduction pathway originating in the sinoatrial node but at a rate exceeding 100 beats per minute. While stress or pain can cause tachycardia, premature ventricular contractions are ectopic beats originating from the ventricles, bypassing the normal atrial conduction system. There is no direct electrophysiological mechanism where ventricular ectopy converts the heart back into a rapid but otherwise normal sinus rhythm originating from the atria.
Choice B rationale
Rapid atrial flutter is a supraventricular tachycardia caused by a macro-reentrant circuit within the atria, typically producing a characteristic sawtooth pattern on an EKG. Premature ventricular contractions occur below the Bundle of His and do not typically trigger atrial re-entrant circuits. While both involve irritability of the cardiac tissue, the pathology of atrial flutter is localized to the upper chambers, whereas frequent ventricular ectopy primarily predisposes the heart to more lethal ventricular-based arrhythmias.
Choice C rationale
Atrioventricular junctional rhythm occurs when the AV node takes over as the primary pacemaker of the heart, usually at a rate of 40 to 60 beats per minute. This typically happens if the sinoatrial node fails or is suppressed. Frequent premature ventricular contractions signify increased ventricular excitability rather than a failure of the upper pacemakers. Therefore, the progression of frequent ventricular ectopy is usually toward faster, more chaotic ventricular rhythms rather than a slower junctional escape rhythm.
Choice D rationale
Ventricular tachycardia is defined as three or more consecutive premature ventricular contractions at a rate greater than 100 beats per minute. Frequent ventricular ectopy indicates significant myocardial irritability. When a premature contraction falls during the vulnerable period of the T-wave, known as the R-on-T phenomenon, it can trigger a sustained run of ventricular tachycardia. This rhythm is life-threatening because it severely reduces cardiac output due to decreased diastolic filling time and loss of atrial kick.
Correct Answer is A
Explanation
Choice A rationale
Advanced age and leukemia significantly compromise the immune system through decreased white blood cell function. A triple-lumen central line provides a direct vascular portal for opportunistic pathogens, increasing the risk of healthcare-associated infections. Leukemia results in neutropenia, where absolute neutrophil counts often fall below 1500 cells/mm, hindering the body's ability to mount an inflammatory response. Consequently, this patient is at the highest statistical risk for developing overwhelming systemic infection and subsequent septic shock.
Choice B rationale
While HIV causes immunodeficiency by depleting CD4+ T-lymphocytes, typically defined as fewer than 500 cells/mm in symptomatic stages, the use of a PEG tube for enteral feeding is a relatively closed system compared to a central venous catheter. While the risk of peritonitis or localized site infection exists, it does not carry the same immediate high-level risk of direct bloodstream seeding as a central line in an elderly, neutropenic patient with a hematologic malignancy.
Choice C rationale
A pediatric patient with an open fracture faces risks related to osteomyelitis or localized tissue infection from environmental contaminants. However, younger patients generally possess more robust physiological reserves and more active immune systems compared to the elderly. Peripheral IVs carry a lower risk of systemic bacteremia than central venous access devices. While serious, an open fracture in a 12-year-old is less likely to progress rapidly to septic shock than a central line infection in leukemia.
Choice D rationale
Pneumonia is a common precursor to sepsis, but a 65-year-old receiving oral antibiotics is likely hemodynamically stable and managing the infection at a localized pulmonary level. The use of oral rather than IV antibiotics suggests a less severe clinical presentation. While the elderly are vulnerable, the absence of profound immunosuppression or invasive central vascular access makes this individual a lower risk for immediate septic shock compared to the patient described in Choice A.
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