Which of the following might be injected into a patient who is prone to forming blood clots and therefore at heart attack or stroke?
Thromboplastin
Fibrinogen
Fibrin
Factor X
Heparin
The Correct Answer is E
A. Thromboplastin: Thromboplastin (tissue factor) initiates the extrinsic coagulation cascade and promotes clot formation; injecting it would increase clotting, not prevent heart attack or stroke.
B. Fibrinogen: Fibrinogen is the soluble precursor that is converted to fibrin during clot formation; giving fibrinogen would support clot formation, not reduce it.
C. Fibrin: Fibrin is the insoluble protein that forms the mesh of a clot; injecting fibrin would directly contribute to clotting.
D. Factor X: Factor X is a key coagulation factor (when activated to Xa it drives thrombin formation); administering it would promote coagulation rather than prevent it.
E. Heparin: Heparin is an anticoagulant that enhances antithrombin activity and reduces clot formation; it is used to prevent or treat thrombosis and reduce risk of heart attack/stroke.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
True: O₂ binds to the heme (iron) portion of hemoglobin, whereas CO₂ binds primarily to amino groups on the globin (forming carbaminohemoglobin) and much CO₂ is also converted to bicarbonate inside RBCs, so they bind/are carried at different sites/forms.
Correct Answer is B
Explanation
A. It is a cause of anemia: Sickle cells are fragile and hemolyze prematurely, producing a chronic hemolytic anemia.
B. It is cause of malaria: Malaria is caused by Plasmodium parasites; sickle-cell disease does not cause malaria (in fact, sickle-cell trait provides some protection against severe malaria).
C. It is due to a hereditary hemoglobin defect: Sickle-cell disease is inherited and results from a mutation in the β-globin (HBB) gene producing hemoglobin S.
D. It is advantageous in that it can protect carriers against malaria: Heterozygous carriers (sickle-cell trait) have relative resistance to severe forms of Plasmodium falciparum malaria.
E. It is caused by excessive allele that modifies the structure of hemoglobin: The disease is caused by a point mutation (a single nucleotide change) in the β-globin allele that alters hemoglobin structure (Glu→Val at position 6). Saying “excessive allele” is misleading -the cause is a specific mutant allele, not an “excessive” one.
Whether you are a student looking to ace your exams or a practicing nurse seeking to enhance your expertise , our nursing education contents will empower you with the confidence and competence to make a difference in the lives of patients and become a respected leader in the healthcare field.
Visit Naxlex, invest in your future and unlock endless possibilities with our unparalleled nursing education contents today
Report Wrong Answer on the Current Question
Do you disagree with the answer? If yes, what is your expected answer? Explain.
Kindly be descriptive with the issue you are facing.