A client with type 1 diabetes who takes insulin reports taking propranolol for hypertension. Why is the nurse concerned?
The beta-blocker can mask the symptoms of hypoglycemia.
Using the two agents together increases the risk of ketoacidosis.
Propranolol increases insulin requirements because of receptor blocking.
The beta-blocker can cause insulin resistance.
The Correct Answer is B
A) While beta-blockers can mask some symptoms of hypoglycemia, the major concern in this scenario is ketoacidosis.
B) Beta-blockers, like propranolol, can mask the typical symptoms of hypoglycemia (tachycardia, palpitations), leading to a delayed recognition of low blood sugar levels.
C) Propranolol may enhance the hypoglycemic effect of insulin, but the primary concern in this case is ketoacidosis.
D) Beta-blockers can indeed cause insulin resistance, but the primary concern in this context is the risk of ketoacidosis.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
a) Taking digoxin at bedtime is not a critical instruction. The important aspect is to take it consistently at the same time each day.
b) A diet high in bran, fiber, and calcium is not specifically related to digoxin administration. Digoxin has specific dietary considerations related to potassium levels.
c) Checking the pulse is crucial for a client taking digoxin, as it helps monitor for potential toxicity. Digoxin can cause bradycardia, and the pulse rate is an essential parameter to assess before administering each dose.
d) While monitoring the heart rate is important, setting a specific threshold (e.g., not taking digoxin if the heart rate exceeds 90 beats per minute) is not a standard recommendation. It's generally more nuanced and individualized.
Correct Answer is B
Explanation
A) Antibiotic resistance is not a reason to adjust the dose of gentamicin.
B) Gentamicin is primarily excreted by the kidneys. In the presence of renal disease, the dose may need to be adjusted to prevent toxicity.
C) Liver disease does not directly impact the metabolism or excretion of gentamicin, so it is not typically a factor in dose adjustment.
D) Interpatient variation alone may not prompt a specific dose adjustment; individual patient factors, especially renal function, are crucial in determining gentamicin dosage.
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