A nurse cares for a patient experiencing diabetic ketoacidosis who presents with Kussmaul respirations. What action would the nurse take?
Administration of IV insulin
Implementation of seizure precautions
Administration of oxygen via face mask
Intravenous administration of 10% glucose
The Correct Answer is A
Choice A reason: IV insulin is essential in diabetic ketoacidosis (DKA) to lower blood glucose by facilitating cellular uptake and halting ketogenesis. Kussmaul respirations indicate metabolic acidosis from ketone accumulation. Insulin corrects hyperglycemia and acid-base imbalance, addressing the root cause of DKA by reducing ketone production and restoring metabolic control, critical for patient stabilization.
Choice B reason: Seizure precautions are not indicated for DKA, as seizures are not a primary complication. Kussmaul respirations result from metabolic acidosis, not neurological issues. Implementing seizure precautions diverts attention from addressing hyperglycemia and acidosis, which require insulin and fluid therapy to correct the metabolic derangements causing rapid breathing and systemic instability.
Choice C reason: Oxygen via face mask is unnecessary unless hypoxemia is confirmed. Kussmaul respirations are a compensatory mechanism for metabolic acidosis, not hypoxia. Oxygen does not address the underlying hyperglycemia or ketosis in DKA. The focus should be on insulin and fluids to correct the metabolic acidosis driving the respiratory pattern.
Choice D reason: IV 10% glucose is inappropriate during initial DKA treatment, as patients have hyperglycemia. Administering glucose could worsen hyperglycemia and osmotic diuresis, exacerbating dehydration and acidosis. Insulin and fluids are needed to lower glucose and correct acidosis, with glucose administration considered only if hypoglycemia occurs during treatment, which is not indicated here.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A reason: Respiratory failure is a concern in peritonitis if abdominal distension impairs diaphragm movement, but it is not the highest priority. Sepsis, from bacterial spread, poses a more immediate life-threatening risk, causing systemic inflammation and shock. Monitoring respiratory status is secondary to addressing the infectious source driving peritonitis complications.
Choice B reason: Diabetes is not a direct complication of peritonitis. While it may complicate management by predisposing to infections, peritonitis itself does not cause diabetes. Sepsis is the primary concern, as bacterial peritonitis can rapidly progress to systemic infection, requiring urgent antibiotics and monitoring to prevent multi-organ failure.
Choice C reason: Sepsis is the highest priority in peritonitis, as bacterial contamination from gastrointestinal perforation or infection can lead to systemic inflammatory response syndrome, shock, and organ failure. Early recognition of fever, tachycardia, and hypotension is critical to initiate antibiotics and fluids, preventing mortality in this life-threatening complication of peritonitis.
Choice D reason: Heart attack is not a primary complication of peritonitis. While sepsis may strain the cardiovascular system, increasing myocardial demand, peritonitis itself does not cause coronary occlusion. Sepsis is the more immediate threat, as it drives systemic inflammation and shock, requiring urgent intervention to prevent progression to multi-organ dysfunction.
Correct Answer is B
Explanation
Choice A reason: Morphine sulfate IV is controversial in pancreatitis, as it may cause sphincter of Oddi spasm, potentially worsening pancreatic duct obstruction and pain. Alternative analgesics like meperidine are preferred. Morphine does not directly promote healing and may complicate pancreatitis management, making it an inappropriate choice for this care plan.
Choice B reason: Maintaining NPO status and administering IV fluids is critical in acute pancreatitis to rest the pancreas, reducing enzyme secretion that exacerbates inflammation. IV fluids correct dehydration from vomiting or third-spacing, supporting hemodynamic stability and promoting healing by minimizing pancreatic stimulation and maintaining perfusion to vital organs.
Choice C reason: Semi-Fowler’s position improves respiratory comfort and reduces abdominal pressure, but it is not the primary intervention for healing in pancreatitis. It aids symptom management, not pancreatic recovery. NPO status and IV fluids directly address the inflammatory process and dehydration, making them more critical for promoting healing in this condition.
Choice D reason: Small, frequent feedings are contraindicated in acute pancreatitis, as oral intake stimulates pancreatic enzyme secretion, worsening inflammation and pain. Patients are kept NPO to rest the pancreas. Feedings may be introduced later in recovery, but during acute phases, this intervention hinders healing and is not included in the care plan.
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