A nurse is caring for a client who is to undergo a bilateral prophylactic mastectomy. The client states that her family opposes her decision. Which of the following responses should the nurse make?

Choice A reason: A quiet environment reduces sensory stimulation, which can elevate intracranial pressure (ICP) by increasing cerebral blood flow. Minimizing noise helps stabilize intracranial dynamics, preventing exacerbation of brain injury. This intervention supports neurological stability, critical in traumatic brain injury management to avoid secondary damage.
Choice B reason: Monitoring vital signs every 8 hours is inadequate for increased ICP, which requires frequent checks (e.g., every 1-2 hours). Changes in blood pressure or respiration signal worsening ICP, risking herniation. Infrequent monitoring delays detection of neurological deterioration, compromising timely intervention in brain injury.
Choice C reason: Coughing and deep breathing increase intrathoracic pressure, elevating ICP by impeding cerebral venous return. This is contraindicated in traumatic brain injury, as it risks worsening cerebral edema or causing herniation, potentially leading to severe neurological damage or fatal outcomes in affected clients.
Choice D reason: A 30-degree head-of-bed elevation promotes cerebral venous drainage, reducing ICP. This position optimizes cerebral perfusion pressure, minimizing venous congestion in traumatic brain injury. It is a critical intervention to prevent secondary brain injury, supporting neurological recovery by stabilizing intracranial dynamics effectively.
Choice E reason: Stool softeners prevent straining during bowel movements, which increases intrathoracic and intracranial pressure. In traumatic brain injury, straining risks exacerbating ICP, potentially causing herniation. This intervention ensures smoother bowel movements, maintaining ICP stability and supporting safe management of brain injury.

Choice A reason: Hyperactive deep tendon reflexes are not linked to morphine toxicity. Morphine, an opioid, depresses the central nervous system, reducing reflexes. Hyperactive reflexes suggest neurological or stimulant effects, not opioid overdose, which primarily causes respiratory and consciousness depression in affected clients.
Choice B reason: Fluid retention is not a primary sign of morphine toxicity. Morphine may cause urinary retention via sphincter tone increase, but fluid overload is unrelated. Toxicity manifests as respiratory depression or sedation, driven by mu-opioid receptor overstimulation, not fluid balance alterations.
Choice C reason: Prolonged QT interval is associated with medications like antiarrhythmics, not morphine. Morphine toxicity primarily causes respiratory depression and sedation via central nervous system effects. Cardiac effects are rare, and QT prolongation is not a hallmark of opioid overdose in clinical settings.
Choice D reason: Bradypnea indicates morphine toxicity, as opioids depress the brainstem’s respiratory center via mu-receptor overstimulation. This slows breathing, risking hypoxia and respiratory arrest, a life-threatening complication requiring immediate intervention like naloxone to reverse opioid effects and restore normal respiratory function.