A nurse is caring for a patient with a confirmed myocardial infarction. The physician orders morphine for pain relief.
What is the primary action of morphine in the management of MI?
It prevents the formation of blood clots in the coronary arteries.
It reduces anxiety and decreases myocardial oxygen demand.
It dilates the coronary arteries and increases oxygen delivery.
It improves blood pressure by causing vasoconstriction.
The Correct Answer is B
Choice A rationale
Morphine does not possess anticoagulant or antiplatelet properties. The prevention of blood clot formation in the coronary arteries is managed by medications such as aspirin, heparin, or clopidogrel. While morphine is essential for comfort, it does not directly interfere with the coagulation cascade or the aggregation of platelets at the site of a ruptured atherosclerotic plaque. Standard laboratory monitoring for anticoagulation includes the activated partial thromboplastin time, which normally ranges from 30 to 40 seconds.
Choice B rationale
Morphine is the gold standard for MI pain because it serves multiple purposes. Beyond its analgesic effects, it acts as a venodilator, which reduces preload and the workload of the heart. By relieving intense pain and the associated anxiety, it blunts the sympathetic nervous system response, leading to a decrease in heart rate and myocardial oxygen consumption. This helps balance the oxygen supply-demand mismatch in the ischemic heart tissue, potentially limiting the size of the infarction.
Choice C rationale
Nitroglycerin is the primary medication used to dilate coronary arteries and increase oxygen delivery to the myocardium. While morphine has some minor vasodilatory effects, its primary benefit in myocardial infarction is not active coronary artery dilation. Morphine focuses more on reducing the oxygen demand of the heart through systemic venous relaxation and sedation. Oxygen saturation should ideally be maintained above 90.
Choice D rationale
Morphine actually causes vasodilation, not vasoconstriction. Therefore, it tends to lower blood pressure rather than improve it. Nurses must be vigilant in monitoring for hypotension after administration. Vasoconstrictors like norepinephrine would be used in cardiogenic shock to increase blood pressure, but they would increase the workload of the heart, which is counterproductive in an uncomplicated myocardial infarction. A normal systolic blood pressure is generally considered to be less than 120 mmHg and greater than 90 mmHg. .
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A rationale
Sinus bradycardia is defined by a heart rate below 60 beats per minute with a regular rhythm and discernible P waves. In this case, the client has a heart rate of 92 per minute and an irregular rhythm, which immediately rules out bradycardia. Furthermore, sinus rhythms must have identifiable P waves and measurable PR intervals, both of which are absent in this client's presentation, pointing toward a more chaotic supraventricular origin.
Choice B rationale
First-degree heart block is characterized by a consistent delay in conduction between the atria and ventricles, resulting in a PR interval greater than 0.20 seconds. However, the rhythm remains regular, and P waves must be clearly visible and associated with every QRS complex. The client in the scenario has an irregular rhythm and unidentifiable P waves, which is inconsistent with the stable, albeit delayed, conduction seen in a first-degree block.
Choice C rationale
Supraventricular tachycardia usually manifests as a very rapid, regular rhythm with rates often exceeding 150 beats per minute. While P waves may be difficult to see because they are buried in the preceding T waves, the hallmark is the absolute regularity of the R-to-R intervals. The client's rhythm is described as irregular, which is the primary clinical feature that distinguishes atrial fibrillation from the regular, rapid pacing of a supraventricular tachycardia.
Choice D rationale
Atrial fibrillation is defined by the absence of discrete P waves and an irregularly irregular ventricular rhythm. The fibrillatory waves from the atria do not produce a measurable PR interval because there is no organized atrial depolarization. The QRS duration of 0.10 seconds is within the normal range of 0.06 to 0.12 seconds, indicating that ventricular conduction is still following the normal pathways once the atrioventricular node allows an impulse through.
Correct Answer is A
Explanation
Choice A rationale
The normal range for serum creatinine is approximately 0.6 to 1.2 mg/dL for adults. A value of 2.1 mg/dL is significantly elevated, indicating a reduction in the glomerular filtration rate and impaired renal function. In the context of a myocardial infarction, low cardiac output can lead to pre-renal azotemia or acute tubular necrosis. This elevation serves as a primary marker for the development of acute kidney injury, reflecting the kidneys' inability to clear metabolic waste effectively.
Choice B rationale
Serum osmolality represents the concentration of particles dissolved in the blood, with a normal range typically between 275 and 295 mOsm/kg H2O. A value of 290 mOsm/kg H2O is within the normal physiological limits. This indicates a stable balance of electrolytes and water in the plasma. Since the value is normal, it does not suggest an increased risk or current state of acute kidney injury or fluid volume disturbances that would damage renal tissue.
Choice C rationale
The normal range for serum magnesium is approximately 1.3 to 2.1 mEq/L. A magnesium level of 2.0 mEq/L is within the normal therapeutic range. While electrolyte imbalances can occur during renal failure, a normal magnesium level does not indicate an increased risk of acute kidney injury. Magnesium is primarily excreted by the kidneys, so while its level might rise if injury occurs, the current value of 2.0 mEq/L indicates that renal clearance is currently sufficient.
Choice D rationale
The normal range for blood urea nitrogen (BUN) is typically 10 to 20 mg/dL. A BUN of 20 mg/dL is at the upper limit of the normal range but is not considered an elevated finding that signifies acute kidney injury on its own. BUN can be influenced by protein intake, hydration status, and liver function. Without an accompanying elevation in creatinine, this normal BUN level does not suggest that the patient is at high risk for kidney failure.
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