Exhibits
The Correct Answer is []
Rationale for Correct Choices:
• Place on Oxygen: The client’s oxygen saturation dropped to 85% on room air, indicating hypoxemia. Supplemental oxygen is required immediately to maintain levels above 93% and prevent worsening respiratory failure. This is consistent with the provider’s orders and emergent asthma care protocols.
• Administer Albuterol: Albuterol is a rapid-acting beta-2 agonist bronchodilator that relieves airway constriction in acute asthma. It directly addresses the wheezing, chest tightness, and severe shortness of breath. Frequent nebulized doses are recommended in status asthmaticus.
• Status Asthmaticus: This is a severe, life-threatening asthma exacerbation unresponsive to initial bronchodilator therapy. The client’s confusion, tachypnea, hypoxemia, and inability to speak in full sentences are classic signs. Without prompt treatment, it can progress to respiratory arrest.
• Pulse Oximetry : Continuous pulse oximetry helps track oxygenation and the effectiveness of interventions. Given the hypoxemia, frequent monitoring is essential to prevent respiratory arrest.
• Respirations Respiratory rate and pattern reflect both asthma severity and the client’s progression toward respiratory fatigue. Worsening asthma can cause sudden respiratory rate drops as exhaustion sets in.
Rationale for Incorrect Choices:
• Suction Airway: There is no evidence of excessive secretions or mucus plugging; the main issue is bronchospasm and airway narrowing, not obstruction from secretions.
• Administer a Diuretic: Diuretics treat fluid overload (e.g., pulmonary edema), which is not present here. This client’s symptoms stem from asthma, not heart failure or volume excess.
• Obtain Sputum Culture: This is done if infection is suspected; the client denies fever and shows no purulent sputum production. This is an acute asthma exacerbation, not pneumonia.
• Anaphylactic Reaction: While both involve airway compromise, there are no signs of urticaria, hypotension, or sudden onset after allergen exposure. This client’s symptoms developed over 2–3 days.
• Pulmonary Edema: Pulmonary edema causes crackles, frothy sputum, and fluid overload symptoms, none of which are present. This client’s lung sounds are wheezy, not wet.
• RSV: RSV is a viral respiratory infection more common in infants and young children; the presentation and history point to asthma, not viral bronchiolitis.
• Troponin: Troponin measures cardiac injury, which is not suspected here. The client’s tachycardia is secondary to respiratory distress, not myocardial infarction.
• Urine Output: While urine output is important in some critical conditions, it is not the primary priority in acute asthma management.
• Temperature: There is no indication of infection or hyperthermia; temperature monitoring is routine but not a key focus in this acute situation.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Rationale:
A. Overriding Aorta: This is one of the four hallmark defects in Tetralogy of Fallot, where the aorta is positioned directly over the ventricular septal defect, allowing oxygen-poor blood to flow into systemic circulation.
B. Right Ventricular Hypertrophy: This occurs due to increased workload from pulmonic stenosis and ventricular septal defect, causing the right ventricle to thicken, and is a key feature of TOF.
C. Left Ventricular aneurysm: This is not associated with Tetralogy of Fallot. It is a localized outpouching or thinning of the left ventricular wall and is unrelated to the structural defects seen in TOF.
D. Pulmonic Stenosis: Narrowing of the pulmonary valve or artery restricts blood flow to the lungs and is one of the defining components of TOF.
E. Ventricular Septal Defect: A large hole between the right and left ventricles allows mixing of oxygenated and deoxygenated blood and is one of the four defects in TOF.
Correct Answer is B
Explanation
Rationale:
A. Vitamin C with iron tablet: While iron supplements may be useful for treating anemia if blood loss has been significant, they do not address the underlying clotting factor deficiency in Hemophilia A. Vitamin C supports tissue healing, but it is not a treatment for this disorder.
B. Factor VIII: Hemophilia A is caused by a deficiency or dysfunction of clotting factor VIII, leading to impaired blood coagulation. The primary treatment is replacing factor VIII through IV infusion to restore normal clotting ability and prevent or control bleeding episodes.
C. Factor IX: Factor IX is used to treat Hemophilia B, also known as Christmas disease, which involves a deficiency in factor IX. It is not appropriate for Hemophilia A since it does not correct the missing clotting factor.
D. Aspirin: Aspirin impairs platelet function and increases bleeding risk. In clients with bleeding disorders such as Hemophilia A, it is contraindicated because it can worsen bleeding tendencies and complicate management.
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