What test is performed on the patient prior to obtaining an ABG?
Chvostek test.
Blumberg test.
Cushing's test.
Allen test.
The Correct Answer is D
Choice A rationale
The Chvostek test assesses for neuromuscular excitability, specifically facial twitching when the facial nerve is tapped. It is used to evaluate for hypocalcemia, not to determine the patency of collateral circulation before an ABG. Performing this test would not provide any relevant information for the safety of obtaining an arterial blood sample.
Choice B rationale
The Blumberg test, also known as rebound tenderness, is used in abdominal assessment to detect peritoneal inflammation. It involves pressing deeply on an area of abdominal tenderness and then quickly releasing the pressure. This test has no relevance to assessing arterial patency or collateral circulation prior to an ABG.
Choice C rationale
The Cushing's test is not a recognized medical test or diagnostic procedure. There is a "Cushing's reflex" which refers to a physiological response to increased intracranial pressure, but this is a clinical sign, not a test performed prior to an ABG. This choice is medically inaccurate in this context.
Choice D rationale
The Allen test is performed prior to obtaining an ABG, especially when considering the radial artery. It assesses the patency of the ulnar artery and ensures adequate collateral blood flow to the hand if the radial artery is compromised during or after the arterial puncture. This prevents potential ischemia to the hand, making it a crucial safety step.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A rationale
Uncompensated respiratory acidosis would exhibit a low pH (normal range 7.35-7.45) and an elevated PaCO2 (normal range 35-45 mmHg), with a bicarbonate level (normal range 22-26 mEq/L) that remains within the normal range. In this scenario, the bicarbonate is elevated, indicating a renal compensatory response has begun.
Choice B rationale
The patient's pH of 7.3 indicates acidosis (normal 7.35-7.45). The PaCO2 of 68 mmHg is elevated (normal 35-45 mmHg), indicating a respiratory origin. The HCO3 of 28 mmol/L is elevated (normal 22-26 mEq/L), signifying that the kidneys are attempting to compensate by retaining bicarbonate. Since the pH is still acidic, but the bicarbonate is responding, it is partially compensated respiratory acidosis.
Choice C rationale
Metabolic acidosis is characterized by a low pH (normal 7.35-7.45) and a low bicarbonate level (normal 22-26 mEq/L). The PaCO2 (normal 35-45 mmHg) would either be normal if uncompensated or low if the respiratory system was compensating. Here, the primary disturbance is respiratory.
Choice D rationale
Metabolic alkalosis presents with an elevated pH (normal 7.35-7.45) and an elevated bicarbonate level (normal 22-26 mEq/L). The PaCO2 (normal 35-45 mmHg) would be normal if uncompensated or elevated if the respiratory system was compensating. This patient's pH is acidic, ruling out alkalosis.
Correct Answer is D
Explanation
Choice A rationale
Cyanosis, a bluish discoloration of the skin and mucous membranes, is a late and ominous sign of hypoxia. It becomes clinically apparent when there are approximately 5 grams per deciliter of desaturated hemoglobin. This indicates a significant reduction in oxygen saturation and severe tissue hypoxia, representing a decompensated physiological state.
Choice B rationale
Bradycardia, a slower than normal heart rate (normal range 60-100 beats per minute for adults), is typically a late sign of hypoxia, particularly in adults. Initially, the body often compensates for hypoxia with tachycardia to increase cardiac output and oxygen delivery. Profound and prolonged hypoxia can depress myocardial function, leading to bradycardia.
Choice C rationale
Hypotension, a low blood pressure (normal range systolic 90-120 mmHg, diastolic 60-80 mmHg), is generally considered a late sign of hypoxia. In the early stages, the sympathetic nervous system may initially increase blood pressure as a compensatory mechanism. However, prolonged or severe hypoxia can lead to myocardial depression and vasodilation, resulting in hypotension.
Choice D rationale
Agitation is an early neurological sign of hypoxia. As oxygen levels decrease, cerebral hypoxia affects brain function, leading to irritability, restlessness, and confusion. The brain is highly sensitive to oxygen deprivation, and these behavioral changes are often among the first indicators of inadequate oxygenation, preceding more overt physiological decompensation.
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