Which finding indicates an adverse effect of fenofibrate?
Hyperglycemia
Increased hemoglobin
Hepatotoxicity
Fever
The Correct Answer is C
A. Hyperglycemia: Fenofibrate primarily affects lipid metabolism and is not commonly associated with causing high blood glucose levels. While monitoring is important in clients with diabetes, hyperglycemia is not a typical adverse effect of this medication.
B. Increased hemoglobin: Fenofibrate does not influence hemoglobin levels. Any changes in hemoglobin are unlikely to be related to fenofibrate therapy and should prompt evaluation for other underlying conditions.
C. Hepatotoxicity: Fenofibrate can cause liver injury, evidenced by elevated liver enzymes or other signs of hepatotoxicity. Monitoring liver function tests before and during therapy is essential to detect adverse effects early and prevent serious liver complications.
D. Fever: Fever is not a recognized adverse effect of fenofibrate. If a client develops fever, other causes such as infection or unrelated inflammatory processes should be investigated rather than attributing it to the medication.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. Treatment for acute myocardial infarction: Apixaban is an oral anticoagulant used to prevent clot formation. It is not indicated as a primary treatment for acute myocardial infarction, although it may be used in certain patients with atrial fibrillation or thromboembolic risk.
B. Prevention of pulmonary embolism: Apixaban is indicated for the prevention and treatment of venous thromboembolism, including deep vein thrombosis and pulmonary embolism. It inhibits factor Xa, reducing the formation of blood clots in high-risk patients.
C. Treatment for chronic hypertension: Apixaban does not lower blood pressure and is not used to treat hypertension. Its anticoagulant effect targets clotting pathways rather than vascular resistance.
D. Prevention of renal failure: Apixaban has no direct effect on kidney function and is not used to prevent renal failure. Its role is limited to anticoagulation and reducing thromboembolic events.
Correct Answer is C
Explanation
A. Decreased inflammation: Vasodilators primarily act on vascular smooth muscle to relax blood vessels, reducing systemic vascular resistance. They do not directly target inflammatory pathways, so reducing inflammation is not their therapeutic effect.
B. Increased heart rate: While some vasodilators may cause reflex tachycardia as a secondary response to lowered blood pressure, an increased heart rate is not the intended therapeutic effect. The primary goal is to reduce vascular resistance and blood pressure.
C. Decreased blood pressure: The main therapeutic effect of vasodilators is the relaxation of arterial and sometimes venous smooth muscle, which lowers systemic vascular resistance. This results in decreased blood pressure, easing the workload on the heart and improving tissue perfusion.
D. Increased platelet count: Vasodilators do not affect platelet production or function. Their mechanism of action is centered on vessel relaxation rather than hematologic changes, so platelet count remains unaffected.
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