A client is admitted to the emergency department with chest pain and shortness of breath. The client states that the chest pain has been happening intermittently all day, lasts about 15 minutes at a time, and has been happening without any physical exertion or emotional stress. STAT lab results reveal troponin levels of 0.01 ng/mL, and a 12-lead electrocardiogram (ECG) shows sinus tachycardia with a T-wave inversion. Which diagnosis would the nurse expect for this client?
NSTEMI
Stable angina
STEMI
Unstable angina
The Correct Answer is D
A. NSTEMI would present with elevated troponin levels indicating myocardial injury, which is not the case here.
B. Stable angina occurs predictably with exertion and is relieved by rest or nitroglycerin.
C. STEMI is characterized by ST-elevation on the ECG and elevated troponin levels, indicating significant myocardial injury.
D. Unstable angina presents with chest pain that occurs at rest or with minimal exertion and is not predictable. The presence of T-wave inversion and normal troponin levels is consistent with this diagnosis.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. Apixaban is an anticoagulant used to prevent clot formation and reduce stroke risk, not specifically to reduce bleeding risk.
B. Rate control is typically achieved with medications like beta-blockers or calcium channel blockers, not anticoagulants like apixaban.
C. Apixaban does not convert heart rhythm; it prevents blood clots.
D. The primary purpose of apixaban in patients with atrial fibrillation is to reduce the risk of stroke by preventing blood clot formation.
Correct Answer is D
Explanation
A. Romiplostim does not increase the production of ADAMTS-13; it is used to increase platelet counts.
B. Romiplostim does not prevent platelet destruction in the spleen; it works by stimulating platelet production.
C. Romiplostim does not prevent platelets from binding with heparin; it is not used for heparin-induced thrombocytopenia.
D. Romiplostim is a thrombopoietin receptor agonist that stimulates the bone marrow to increase platelet production, which is its primary mechanism of action in treating ITP.
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