A laboring client is at 10 cm dilation.
What would the nurse do next?

Choice A rationale

Racemic epinephrine nebulizer therapy is a highly effective measure for bronchodilation in a child with croup. This alpha-adrenergic agonist works by stimulating receptors in the smooth muscle of the airway. This stimulation leads to vasoconstriction, reducing the mucosal edema and swelling of the vocal cords and trachea that are characteristic of croup. The rapid reduction in upper airway inflammation and swelling directly alleviates stridor and respiratory distress, improving airflow and oxygenation.

Choice B rationale

Teaching a child to take long, slow breaths is generally a technique used for anxiety or to prevent hyperventilation. However, in a child with croup, the upper airway obstruction makes deep breathing difficult and may increase their distress. The effort to take long, slow breaths against a narrowed airway can further exacerbate the child's respiratory effort and may not provide any significant relief from the underlying swelling.

Choice C rationale

Administering an oral analgesic would be ineffective for aiding bronchodilation in a child with croup. The primary issue in croup is upper airway inflammation and edema, not pain. Analgesics, such as acetaminophen or ibuprofen, primarily work to reduce fever and discomfort. They have no direct pharmacological effect on the smooth muscles of the bronchi or on the mucosal swelling that is causing the respiratory distress.

Choice D rationale

Urging a child to continue to take oral fluids is important for hydration, but it does not directly aid in bronchodilation. In a child with significant respiratory distress from croup, encouraging oral fluids may actually pose a risk for aspiration due to their difficulty breathing and potential fatigue. While hydration is a supportive measure, it does not address the underlying pathology of airway swelling and is not a primary intervention for bronchodilation.

Choice A rationale

Increasing weight loss is an incorrect characteristic. Nephrotic syndrome is marked by massive proteinuria, which leads to a decrease in plasma oncotic pressure. This fluid shift from the intravascular space to the interstitial space results in significant fluid retention, causing weight gain and edema, not weight loss. The body holds onto fluid, masking any potential muscle or fat wasting.

Choice B rationale

Increased urinary output is not a characteristic of nephrotic syndrome. The condition is associated with severe fluid retention and decreased plasma volume, leading to oliguria, or a decreased urinary output. The kidneys are not effectively filtering protein and fluid is being retained in the body, which directly reduces the amount of fluid that can be excreted as urine.

Choice C rationale

Generalized edema is the most common and striking characteristic of nephrotic syndrome. The massive loss of protein, particularly albumin, in the urine leads to a significant decrease in serum albumin levels. Albumin is crucial for maintaining plasma oncotic pressure. The resulting decrease in oncotic pressure causes fluid to shift from the bloodstream into the interstitial spaces, resulting in widespread or anasarca edema.

Choice D rationale

While hypertension can occur, it is not the most common characteristic associated with nephrotic syndrome. The primary physiological change is the massive proteinuria leading to hypoproteinemia and subsequent edema. Hypertension may develop as a result of volume overload, but it is not a hallmark sign. The most prominent and defining symptom is the severe edema.