A nurse is caring for a client who takes lisinopril for hypertension. Which of the following client statements indicates an adverse effect of the medication?
“I seem to be bruising more easily,"
"I have to urinate frequently."
"I have a nagging, dry cough."
"I have a heightened sense of taste."
The Correct Answer is C
A. “I seem to be bruising more easily,”: Easy bruising is not a typical adverse effect of lisinopril. Bruising may indicate a hematologic issue or another medication effect, but it is unrelated to ACE inhibitor therapy.
B. "I have to urinate frequently.": Increased urination is not commonly associated with lisinopril. Diuretics, rather than ACE inhibitors, are more likely to cause polyuria. This statement does not indicate an adverse effect of lisinopril.
C. "I have a nagging, dry cough.": A persistent, dry cough is a well-known adverse effect of ACE inhibitors like lisinopril. It occurs due to the accumulation of bradykinin in the respiratory tract, and it can be bothersome enough to require medication adjustment or substitution.
D. "I have a heightened sense of taste.": Altered taste perception is not a common adverse effect of lisinopril. While some medications can affect taste, this is not characteristic of ACE inhibitors and is unlikely to be related to the client’s current therapy.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["B","C","F"]
Explanation
A. Administer an enema to promote bowel evacuation: Enemas increase intraluminal pressure and can worsen inflammation or risk perforation in acute diverticulitis. The bowel should be rested during the acute phase to reduce irritation and allow healing. Using an enema can aggravate symptoms and is contraindicated.
B. Prepare to insert a nasogastric tube: The client has nausea, vomiting, abdominal distention, and worsening pain, which can indicate possible ileus or obstruction associated with acute diverticulitis. An NG tube may be needed to decompress the stomach and prevent further vomiting. Preparing for NG tube insertion supports symptom management and prevents complications.
C. Ensure the client is on bed rest: Bed rest decreases bowel activity and reduces discomfort during acute diverticulitis episodes. It helps limit movement that can exacerbate abdominal pain and reduces metabolic demands while the inflammation is being treated.
D. Administer a laxative to prevent constipation: Laxatives stimulate bowel motility, which increases pressure within the colon and can worsen inflammation or risk perforation in acute diverticulitis. These medications are avoided during the acute phase to prevent exacerbation of symptoms or complications.
E. Provide the client with foods that are high in fiber: High-fiber foods help prevent future diverticulitis episodes but are not appropriate during an acute flare. Fiber increases bowel workload and irritation, worsening pain and inflammation. Dietary fiber is introduced only after acute symptoms have resolved.
F. Ensure client is NPO: Keeping the client NPO rests the bowel and reduces stimulation during acute inflammation. NPO status also prepares the client for potential procedures and reduces the risk of worsening symptoms from food intake. This is standard treatment during acute diverticulitis until symptoms improve.
G. Position client in high-Fowler's position: High-Fowler’s position is more useful for clients with respiratory compromise rather than abdominal inflammation. Although sitting upright may provide minor comfort, it does not specifically support management of acute diverticulitis. Supine or semi-Fowler's positions are better tolerated to reduce abdominal strain.
Correct Answer is []
Explanation
Rationale for Correct Choices
• Hypovolemic shock: The client had rapid diuresis after receiving 80 mg IV furosemide, resulting in an output of almost 1 L of urine within 1 hour, followed by a sudden drop to only 30 mL at 0100. The fall in blood pressure from 175/88 to 122/75, rising heart rate, dizziness, thirst, and weak pulses indicate low circulating volume consistent with volume depletion.
• Elevate the client’s feet: Raising the lower extremities promotes venous return to improve cardiac output when circulating volume is reduced. This position can temporarily enhance perfusion to vital organs in early hypovolemia. It is also a non-invasive measure providing symptomatic improvement while other treatments are initiated.
• Administer IV fluids: Excess diuresis from furosemide can lead to acute intravascular depletion, and restoring volume with isotonic fluids helps improve preload and blood pressure. The marked drop in urine output to 30 mL indicates compromised renal perfusion, which requires prompt volume replacement. Improved volume status also stabilizes heart rate and reduces symptoms such as thirst and dizziness.
• Pulse pressure: Narrowing pulse pressure is a key indicator of worsening hypovolemia because falling systolic pressure and compensatory vasoconstriction reduce the difference between systolic and diastolic values. Monitoring trends helps evaluate response to fluid resuscitation. Improvement suggests stabilization of intravascular volume and cardiac output.
• Mental status: Cerebral perfusion is highly sensitive to changes in blood volume, so declining alertness or increasing restlessness may indicate deterioration. Monitoring cognitive status provides early warning of inadequate perfusion or progressing shock. Improvement with treatment reflects recovery of effective circulation.
Rationale for Incorrect Choices
• Septic shock: The client has no fever, elevated WBC count, or infectious symptoms, and the rapid fluid loss following diuresis is a more direct explanation for the decline. The stable temperature and clear lungs further reduce suspicion for infection-related hypotension.
• Cardiogenic shock: The lungs are clear, respiratory rate has normalized, and the initial diuretic improved symptoms, making pump failure less likely. The pattern of high urine output followed by a sharp decline aligns more with fluid depletion than primary cardiac dysfunction.
• Obstructive shock: There are no signs of conditions such as pulmonary embolism or tension pneumothorax, and the vital signs improved rather than deteriorated after diuresis. Clear lung sounds and absence of chest pain argue against mechanical obstruction.
• Administer 1 unit of packed RBCs: There is no evidence of bleeding or anemia, and hemoglobin levels are not provided to justify transfusion. The client’s symptoms align with volume loss from diuresis, not red-cell deficiency.
• Obtain a lactate level: While lactate can help evaluate tissue perfusion, it is not the priority intervention when hypovolemia from medication-induced diuresis is evident. Clinical signs already pinpoint fluid loss as the cause, making fluids and positioning more urgent.
• Administer IV antibiotics: No indicators of infection are present, and antibiotic therapy does not address the current hemodynamic issue. The temperature and assessment findings show no infectious focus requiring treatment.
• Blood culture results: Blood cultures assist in diagnosing sepsis, but there is no clinical suspicion of infection in this scenario. The cause of hypotension is more clearly linked to recent diuresis rather than bacteremia.
• Platelet count: Platelet levels are not relevant to diagnosing or managing hypovolemic shock caused by fluid loss. Platelets would be significant in bleeding disorders, which are not indicated here.
• Temperature: The client’s temperature is stable, and changes would not provide insight into fluid volume status. Temperature monitoring is more appropriate when infection or inflammatory causes are suspected.
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