Exhibits
The nurse suspects that the client may have anemia.
For each statement, click to specify whether the statement is consistent with iron deficiency anemia, vitamin B12 deficient anemia, or folic acid deficient anemia. Each category may support more than one deficiency, but each deficiency must have at least one response selected.
Decreased hemoglobin and hematocrit levels
Uptake often impeded by medications
Result of dietary deficiency
Often associated with chronic alcoholism
Can be caused by malabsorption syndrome
The Correct Answer is {"A":{"answers":"A,B,C"},"B":{"answers":"A,B,C"},"C":{"answers":"A,B,C"},"D":{"answers":"A,C"},"E":{"answers":"A,B,C"}}
Decreased hemoglobin and hematocrit levels:
- Folic acid deficiency anemia: Yes, folic acid deficiency can result in low hemoglobin and hematocrit levels as folate is necessary for red blood cell (RBC) production.
- Iron deficiency anemia: Yes, iron deficiency leads to decreased hemoglobin and hematocrit levels because iron is crucial for hemoglobin synthesis.
- Vitamin B12 deficiency anemia: Yes, B12 deficiency can cause decreased hemoglobin and hematocrit levels due to impaired RBC production.
Rationale: All three forms of anemia can result in low hemoglobin and hematocrit levels due to impaired red blood cell production.
Uptake often impeded by medications:
- Folic acid deficiency anemia: Yes, certain medications, like anticonvulsants or methotrexate, can interfere with folic acid absorption and utilization.
- Iron deficiency anemia: Yes, some medications like proton pump inhibitors (PPIs) or antacids can interfere with iron absorption.
- Vitamin B12 deficiency anemia: Yes, medications such as proton pump inhibitors, H2 blockers, and metformin can interfere with B12 absorption.
Rationale: Medications can affect the absorption of all three nutrients—folic acid, iron, and B12—and lead to deficiencies, especially in individuals taking these medications long-term.
Result of dietary deficiency:
- Folic acid deficiency anemia: Yes, inadequate dietary intake of folate can lead to deficiency and anemia. Common in those with poor diets or increased demand (e.g., pregnancy).
- Iron deficiency anemia: Yes, iron deficiency is commonly caused by inadequate dietary intake of iron-rich foods (e.g., red meat, leafy greens).
- Vitamin B12 deficiency anemia: Yes, insufficient dietary intake, particularly in vegetarians or vegans who avoid animal products, can lead to B12 deficiency.
Rationale: All three anemias can be caused by inadequate dietary intake of the respective nutrients.
Often associated with chronic alcoholism:
- Folic acid deficiency anemia: Yes, chronic alcohol use can impair folic acid absorption and utilization, contributing to deficiency.
- Iron deficiency anemia: Less commonly associated with alcoholism, though heavy drinking can affect iron absorption and cause gastrointestinal bleeding, leading to iron loss.
- Vitamin B12 deficiency anemia: Yes, alcohol use can interfere with vitamin B12 absorption and contribute to deficiency.
Rationale: Chronic alcohol use is often associated with folic acid and B12 deficiencies due to impaired absorption, while its association with iron deficiency is less direct but can occur due to GI bleeding or poor nutrition.
Can be caused by malabsorption syndrome:
- Folic acid deficiency anemia: Yes, conditions like celiac disease or Crohn's disease can impair folate absorption.
- Iron deficiency anemia: Yes, malabsorption syndromes (e.g., celiac disease, Crohn's) can prevent proper iron absorption.
- Vitamin B12 deficiency anemia: Yes, malabsorption syndromes like pernicious anemia or celiac disease can impair B12 absorption in the intestines.
Rationale: All three types of anemia can result from malabsorption syndromes due to difficulty absorbing nutrients from the digestive tract.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["B","C","E","F"]
Explanation
A. Nonsmoker: Smoking does not directly contribute to the development of gout. While smoking is a risk factor for many health conditions, it is not a major contributor to the elevated uric acid levels associated with gout.
B. Obesity: Obesity is a well-established risk factor for gout because it leads to increased production of uric acid and reduced renal excretion of uric acid. Higher body fat increases the likelihood of developing hyperuricemia (elevated uric acid levels in the blood), which can form crystals in the joints, leading to gout.
C. Drinks beer nightly: Alcohol, particularly beer, is a significant risk factor for gout. Beer contains high levels of purines, which are broken down into uric acid in the body. Chronic alcohol consumption, especially beer, increases uric acid levels, contributing to the formation of uric acid crystals in the joints, which leads to gout attacks.
D. Daily aspirin: Aspirin, particularly at low doses, can reduce the excretion of uric acid by the kidneys, which can increase the risk of hyperuricemia and gout. However, this effect is typically observed with chronic, low-dose aspirin use, which is not indicated here in the patient's history.
E. Type 2 diabetes mellitus: Type 2 diabetes mellitus is associated with insulin resistance, which impairs the kidney's ability to excrete uric acid. As a result, uric acid builds up in the bloodstream, increasing the risk of gout. Additionally, people with type 2 diabetes often have comorbidities (such as obesity) that further increase the risk of gout.
F. Sleep apnea: Obstructive sleep apnea is associated with intermittent hypoxia (lack of oxygen), which can contribute to the production of uric acid and decreased renal clearance. This increase in uric acid levels raises the risk of hyperuricemia and subsequently gout. Sleep apnea can also be related to metabolic syndrome, which is another risk factor for gout.
Correct Answer is C
Explanation
A. Clear, dark amber-colored urine may indicate dehydration or concentrated urine, which does not necessarily signify improvement in liver function or treatment efficacy.
B. A prothrombin time within normal limits may indicate improved liver function; however, it is not the primary goal of the treatment plan focused on managing ascites and fluid retention in cirrhosis.
C. Decreased abdominal girth is a key indicator of progress in managing fluid retention associated with cirrhosis, as the treatment plan aims to reduce ascites through a low sodium diet and albumin infusions.
D. Improved level of consciousness is essential for overall recovery but is not the primary measure of progress related to fluid management and treatment effects in this context.
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