How does montelukast (Singulair), a leukotriene blocker, work in the treatment of asthma?
Increased vessel permeability
Reduced bronchoconstriction and mucus secretion
Increased inflammation
Increased bronchoconstriction
The Correct Answer is B
Choice A reason: Montelukast does not increase vessel permeability. It blocks the effects of leukotrienes, which are inflammatory mediators that cause vessel permeability, bronchoconstriction, and mucus secretion. By blocking leukotrienes, montelukast reduces vessel permeability and inflammation.
Choice B reason: Montelukast reduces bronchoconstriction and mucus secretion. It blocks the effects of leukotrienes, which are inflammatory mediators that cause bronchoconstriction and mucus secretion. By blocking leukotrienes, montelukast improves airflow and reduces asthma symptoms.
Choice C reason: Montelukast does not increase inflammation. It blocks the effects of leukotrienes, which are inflammatory mediators that cause inflammation, bronchoconstriction, and mucus secretion. By blocking leukotrienes, montelukast reduces inflammation and prevents asthma exacerbations.
Choice D reason: Montelukast does not increase bronchoconstriction. It blocks the effects of leukotrienes, which are inflammatory mediators that cause bronchoconstriction, mucus secretion, and inflammation. By blocking leukotrienes, montelukast reduces bronchoconstriction and improves airflow.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
Choice A reason: This is the correct mechanism of action of propranolol. Propranolol is a beta blocker medication, which means that it blocks the beta receptors in the heart and blood vessels. This reduces the sympathetic stimulation in cardiac muscle, which lowers the heart rate, contractility, and oxygen demand. This also reduces the renin release from the kidneys, which lowers the blood pressure.
Choice B reason: This is not the mechanism of action of propranolol. Propranolol does not increase the diuretic response in the renal tubules. This is the effect of diuretic medications, such as furosemide or hydrochlorothiazide, which increase the urine output and decrease the blood volume and pressure.
Choice C reason: This is not the mechanism of action of propranolol. Propranolol does not inhibit the conversion of angiotensin I to angiotensin II. This is the effect of angiotensin-converting enzyme (ACE) inhibitors, such as lisinopril or enalapril, which block the formation of angiotensin II, a potent vasoconstrictor that raises the blood pressure.
Choice D reason: This is not the mechanism of action of propranolol. Propranolol does not block alpha receptors throughout the body. This is the effect of alpha blockers, such as doxazosin or prazosin, which block the alpha receptors in the blood vessels and cause vasodilation, which lowers the blood pressure.
Correct Answer is B
Explanation
Choice A reason: This is not what will happen when a person is hypotensive. Baroreceptors are sensory receptors that detect changes in blood pressure. When a person is hypotensive, the baroreceptors are more active, not less, and they send signals to the brain to increase the blood pressure.
Choice B reason: This is what will happen when a person is hypotensive. SNS stands for sympathetic nervous system, which is the part of the autonomic nervous system that prepares the body for fight or flight response. When a person is hypotensive, the SNS is activated to increase the heart rate, contractility, and vasoconstriction, which all raise the blood pressure.
Choice C reason: This is not what will happen when a person is hypotensive. Person will be bradycardic means that the person will have a slow heart rate, usually below 60 beats per minute. When a person is hypotensive, the opposite will happen, as the heart rate will increase to compensate for the low blood pressure.
Choice D reason: This is not what will happen when a person is hypotensive. SNS is suppressed means that the sympathetic nervous system is inhibited or reduced in activity. When a person is hypotensive, the SNS is not suppressed, but rather stimulated, to increase the blood pressure.
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