The client with chronic renal failure has a glomerular filtration rate (GFR) of 40 mL/min/1.73 m², as measured by a 24-hour creatinine clearance. What is the nurse's interpretation of this finding?
Increased glomerular filtration rate, client at risk for fluid overload
Reduced glomerular filtration rate, client at risk for fluid overload
Reduced glomerular filtration rate, client at risk for dehydration
Increased glomerular filtration rate, client at risk for dehydration
The Correct Answer is B
Choice A reason: A GFR of 40 mL/min/1.73 m² is below normal (≥90), indicating reduced, not increased, kidney function. Chronic renal failure impairs fluid excretion, leading to fluid overload, not dehydration. Increased GFR would suggest hyperfiltration, seen in early diabetes, not chronic renal failure, making this interpretation incorrect.
Choice B reason: A GFR of 40 mL/min/1.73 m² indicates reduced kidney function (Stage 3B chronic kidney disease). The kidneys’ impaired ability to excrete fluid increases the risk of fluid overload, causing edema, hypertension, or pulmonary edema. This aligns with the pathophysiology of chronic renal failure, making this the correct interpretation.
Choice C reason: A GFR of 40 mL/min/1.73 m² reflects reduced kidney function, not dehydration risk. Chronic renal failure leads to fluid retention due to decreased filtration, causing hypervolemia, not hypovolemia. Dehydration is associated with prerenal causes, not established chronic kidney disease, making this interpretation inconsistent with the clinical scenario.
Choice D reason: A GFR of 40 mL/min/1.73 m² is reduced, not increased, in chronic renal failure. Reduced GFR leads to fluid retention, not dehydration, as the kidneys cannot excrete excess fluid. Increased GFR might cause dehydration in rare hyperfiltration states, but this does not apply to chronic renal failure’s pathophysiology.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
Choice A reason: Polyuria, excessive urine output, typically leads to potassium loss, causing hypokalemia, not hyperkalemia. In conditions like diabetes insipidus, increased urination reduces serum potassium as the kidneys excrete more fluid and electrolytes. Elevated potassium is more associated with reduced renal excretion, as in kidney failure, not polyuria.
Choice B reason: Creatinine levels increase in acute kidney injury due to reduced glomerular filtration, impairing the kidneys’ ability to clear creatinine, a muscle metabolism byproduct. Decreased creatinine levels are rare and may reflect low muscle mass, not kidney injury, making this statement incorrect as it contradicts the pathophysiology of renal impairment.
Choice C reason: Specific gravity is increased in hypovolemia, as the kidneys conserve water, producing concentrated urine (high specific gravity, >1.020). Decreased specific gravity occurs in conditions like diabetes insipidus, where dilute urine is produced. This statement is incorrect, as hypovolemia leads to higher, not lower, urine specific gravity.
Choice D reason: Blood urea nitrogen (BUN) increases in dehydration due to reduced renal perfusion, causing the kidneys to reabsorb urea to conserve water. This elevates serum BUN levels, often with a normal creatinine, reflecting prerenal azotemia. This statement correctly aligns with the pathophysiology of dehydration’s effect on renal laboratory values.
Correct Answer is C
Explanation
Choice A reason: Starting an IV infusion of lactated Ringer’s supports fluid status and may be needed for anticoagulation administration in pulmonary embolism. However, it does not address the immediate hypoxemia caused by the embolism’s ventilation-perfusion mismatch. Oxygen therapy is the priority to correct low oxygen levels and prevent tissue hypoxia.
Choice B reason: Morphine IV may relieve pain and anxiety in pulmonary embolism, reducing oxygen demand. However, it does not directly address hypoxemia, the primary life-threatening issue. The ABCDE approach prioritizes breathing, making oxygen therapy the first intervention to stabilize the patient before pain management is considered.
Choice C reason: Pulmonary embolism causes a ventilation-perfusion mismatch, reducing oxygen delivery to the blood, leading to hypoxemia. Administering oxygen therapy immediately increases alveolar oxygen, improving arterial PaO2 and preventing tissue hypoxia. In the ABCDE approach, breathing is prioritized, making oxygen therapy the first intervention to stabilize the client.
Choice D reason: Cardiac monitoring assesses for arrhythmias or right heart strain in pulmonary embolism, which is important for ongoing management. However, it does not correct the immediate threat of hypoxemia. Oxygen therapy addresses the critical reduction in oxygen saturation, taking precedence in the ABCDE approach over monitoring in acute management.
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