The nurse is admitting a client diagnosed with mitral valve regurgitation. Which medication would help decrease afterload for this client?

a. Monitor urine output: Monitoring urine output is important in clients with heart failure to assess renal perfusion and renal function. Decreased urine output may indicate worsening heart failure or inadequate response to treatment. Therefore, monitoring urine output is appropriate in this situation to assess the client's fluid status and response to therapy.

b. Assess pulse oximetry: Assessing pulse oximetry is essential to evaluate the client's oxygen saturation levels, especially in a client presenting with dyspnea and crackles throughout bilateral lung fields. Decreased oxygen saturation may indicate respiratory compromise or worsening heart failure. Therefore, assessing pulse oximetry is necessary to monitor the client's respiratory status and guide interventions.

c. Check PT/INR: Checking PT/INR (Prothrombin Time/International Normalized Ratio) is not indicated in this situation. PT/INR monitoring is typically performed in clients on anticoagulant therapy such as warfarin to assess coagulation status and therapeutic levels. It is not directly relevant to the client's current presentation of dyspnea, tachycardia, and crackles.

d. Infuse 0.9% NS with 20 mEq KCl at 50 ml/hr: Infusing normal saline (0.9% NS) with potassium chloride (KCl) is not appropriate based solely on the client's presentation of dyspnea, anxiety, tachycardia, and crackles. While fluid and electrolyte balance are important considerations in heart failure management, administering IV fluids and electrolytes should be guided by the client's fluid status, electrolyte levels, and response to treatment.

e. Administer 40 mg furosemide IV now: Administering furosemide IV (intravenous loop diuretic) is appropriate in this situation to manage the client's symptoms of dyspnea and crackles indicative of fluid overload in heart failure. Furosemide helps promote diuresis and reduce fluid volume, thereby alleviating symptoms of congestion and improving respiratory distress.

Rheumatic fever: This inflammatory condition can affect various parts of the heart, including the mitral and aortic valves. Repeated episodes of rheumatic fever can lead to scarring and thickening of the heart valves, eventually causing stenosis (narrowing) of the mitral or aortic valve.

b. Endocarditis: This infection of the heart lining or valves can damage the structures, but it's not as directly linked to both mitral and aortic stenosis as rheumatic fever.

c. Marfan’s syndrome:Marfan syndrome is a genetic disorder that can affect connective tissue throughout the body, including the heart valves. It is commonly associated with aortic root dilatation and aortic regurgitation rather than aortic stenosis. While aortic valve involvement can occur in Marfan syndrome, it is not typically associated with mitral stenosis.

d. Female gender: While gender differences exist in the prevalence of certain cardiovascular conditions, such as rheumatic heart disease affecting more females than males, gender alone is not a significant risk factor for the development of either mitral or aortic stenosis.