The nurse is providing education for a patient diagnosed with Myasthenia Gavis (MG) taking pyridostigmine. The nurse understands teaching has been effective when the patient states. "I will....
be able to crush the sustained release tablet because of my difficulty swallowing
take pyridostigmine 30-60 minutes before meals to improve muscle function for chewing and swallowing
use pyridostigmine only as needed to relieve symptoms of muscle weakness and fatigue.
double a dose if I have increased fatigue to increase the effects of the medication."
The Correct Answer is B
A. Crushing sustained-release tablets is not recommended because doing so could interfere with the controlled release of the medication, which is designed to release slowly over time. This could lead to adverse effects.
B. Pyridostigmine is used to improve muscle strength in patients with Myasthenia Gravis (MG). Taking it 30-60 minutes before meals helps maximize its effect on muscle strength, particularly for activities like chewing and swallowing, which are often affected by MG.
C. Pyridostigmine is typically used regularly, not just as needed. Consistent dosing helps maintain muscle strength and manage the symptoms of MG, rather than only addressing occasional weakness and fatigue.
D. Doubling the dose without medical advice can be dangerous. The prescribed dose should be followed, and any changes should be discussed with a healthcare provider to avoid potential overdose or side effects.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. is experiencing a respiratory infection and is short of breath: While a respiratory infection and shortness of breath can exacerbate symptoms of Myasthenia Gravis (MG), this condition does not specifically increase the risk of a cholinergic crisis. However, respiratory issues can worsen during a crisis, but they are not the main trigger.
B. reports taking a daily extra dose of their anticholinesterase medication: Cholinergic crisis occurs when there is an overdose of anticholinesterase medication, which increases acetylcholine levels and causes overstimulation of the parasympathetic nervous system. Symptoms include excessive salivation, sweating, diarrhea, bradycardia, and respiratory distress. Taking an extra dose of anticholinesterase medication can lead to this crisis, making the patient most at risk in this scenario.
C. has a family history of autoimmune disorders: While a family history of autoimmune disorders might indicate a predisposition to developing autoimmune conditions, it does not directly increase the risk of a cholinergic crisis. Cholinergic crises are more specifically related to medication dosing in MG.
D. has a past medical history of type 2 diabetes mellitus: Type 2 diabetes mellitus does not have a direct correlation with an increased risk of developing a cholinergic crisis in a patient with MG. The risk of cholinergic crisis is more related to the administration of anticholinesterase medications, not diabetes.
Correct Answer is C
Explanation
A. GI & cerebral blood flow: the body’s overall blood flow is important, myocardial oxygen consumption is more directly related to the heart's workload. GI and cerebral blood flow do not have a direct impact on myocardial oxygen demand.
B. Renal blood flow: Renal blood flow does not directly affect myocardial oxygen consumption. The heart's oxygen needs are more influenced by factors such as heart rate, contractility, preload, and afterload rather than the blood flow to the kidneys.
C. Heart rate & contractility: Myocardial oxygen consumption increases as the heart rate and contractility increase. Both heart rate (the number of beats per minute) and contractility (the strength of each contraction) require more oxygen to function. As these factors increase, the heart has to work harder, thus consuming more oxygen.
D. Cerebral blood flow: While cerebral blood flow is essential for brain function, it does not directly influence myocardial oxygen consumption. The heart’s oxygen needs are more closely linked to factors that affect the heart's workload, such as preload, afterload, heart rate, and contractility.
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