(1 point). Listen.
If a patient has dysfunction in the normal secretion of natriuretic hormones, which laboratory result should the nurse monitor?
Phosphorus.
Calcium.
Sodium.
Aldosterone.
The Correct Answer is C
Choice A rationale:
Phosphorus. Rationale: Dysfunction in the normal secretion of natriuretic hormones is more likely to affect sodium and water balance, not phosphorus. Phosphorus levels are regulated by other mechanisms in the body and may not be directly influenced by natriuretic hormones. Choice A is incorrect.
Choice B rationale:
Calcium. Rationale: Calcium levels are not typically influenced by natriuretic hormones. Calcium regulation is primarily under the control of parathyroid hormone (PTH) and calcitonin. Dysfunction in natriuretic hormone secretion would not directly impact calcium levels. Choice B is incorrect.
Choice C rationale:
Sodium. Rationale: Natriuretic hormones, such as atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP), play a crucial role in regulating sodium balance in the body. If a patient has dysfunction in the normal secretion of these hormones, it can lead to sodium imbalance, potentially causing sodium retention and fluid overload. Therefore, monitoring sodium levels is essential in such cases. Choice C is the correct answer.
Choice D rationale:
Aldosterone. Rationale: Aldosterone is a hormone that primarily regulates sodium and potassium balance in the body. Dysfunction in natriuretic hormone secretion is not directly related to aldosterone. Therefore, monitoring aldosterone levels would not be the most appropriate action in this context. Choice D is incorrect. .
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A rationale:
"Chorea" is not related to the pathophysiology of acute rheumatic fever. Chorea is a movement disorder, but it is not a term associated with the immune response to group A streptococcal cell membrane antigens.
Choice B rationale:
"M proteins" are the key component involved in the pathophysiology of acute rheumatic fever. Group A streptococcal M proteins trigger an abnormal immune response that can lead to acute rheumatic fever and its associated complications, including rheumatic heart disease.
Choice C rationale:
"C-reactive protein" is not the primary antigen or component involved in the pathophysiology of acute rheumatic fever. While C-reactive protein may increase in response to inflammation, it is not the main factor responsible for the development of this condition.
Choice D rationale:
"Streptolysin O" is not the primary antigen involved in the pathophysiology of acute rheumatic fever. Streptolysin O is a toxin produced by group A streptococci, but it is not the antigen responsible for the abnormal immune response leading to acute rheumatic fever. .
Correct Answer is D
Explanation
Choice A rationale:
Decreases in the supply or an increase in demand for sodium are not the primary components in the development of acute coronary syndromes. Acute coronary syndromes primarily involve the blood supply to the heart muscle, and sodium levels are not directly related to this condition.
Choice B rationale:
Glucose levels do not play a central role in the development of acute coronary syndromes. While glucose metabolism is important for the heart's energy needs, acute coronary syndromes are primarily related to changes in blood flow and oxygen supply to the heart.
Choice C rationale:
Myoglobin is not the most important component in the development of acute coronary syndromes. Myoglobin is a protein found in muscle tissue, including the heart, but its release into the bloodstream is not specific to acute coronary syndromes. It can be elevated in various conditions, including muscle injury.
Choice D rationale:
The most important component in the development of acute coronary syndromes is a decrease in the supply or an increase in demand for oxygen. Acute coronary syndromes, such as myocardial infarction (heart attack), are typically caused by a disruption in the blood supply to the heart muscle, leading to ischemia and, ultimately, tissue damage. This results in an increased demand for oxygen while the supply is compromised, which can lead to chest pain, tissue injury, and potentially life-threatening consequences. .
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