A 65-year-old patient with a history of Peripheral Artery Disease (PAD) presents to the clinic with complaints of intermittent claudication in the right leg. Which of the following management strategies should the nurse prioritize to improve the patient’s condition?
Encourage the patient to engage in regular low-impact exercise.
Advise the patient to limit fluid intake to prevent edema.
Instruct the patient to wear tight-fitting shoes to improve circulation.
Recommend strict bed rest to minimize pain.
The Correct Answer is A
Choice A reason: Low-impact exercise, like walking, enhances collateral circulation in PAD, increasing blood flow to ischemic muscles. It reduces claudication by stimulating angiogenesis and improving endothelial function, a cornerstone of evidence-based management to alleviate symptoms effectively.
Choice B reason: Limiting fluid intake addresses edema, not a primary PAD issue, which involves arterial insufficiency, not venous stasis. This strategy lacks scientific support for claudication relief, as hydration status doesn’t directly influence arterial perfusion.
Choice C reason: Tight shoes restrict circulation, worsening PAD by compressing arteries and exacerbating ischemia. Proper footwear is essential, but constriction contradicts vascular physiology, potentially increasing pain and tissue damage rather than improving blood flow.
Choice D reason: Bed rest reduces activity, promoting stasis and deconditioning in PAD, worsening claudication. Immobility decreases muscle pump action and collateral flow, conflicting with evidence that exercise improves symptoms, making this detrimental to recovery.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is C
Explanation
Choice A reason: Wheezing indicates airway narrowing, typical in asthma or COPD, not pneumonia. Pneumonia causes alveolar fluid, producing crackles, so diffuse wheezing doesn’t align with its pathophysiology of consolidation.
Choice B reason: Finger clubbing and pallor suggest chronic hypoxia or anemia, not acute pneumonia. These develop over time, whereas pneumonia presents with acute respiratory signs like crackles, not chronic markers.
Choice C reason: Crackles or rales occur in pneumonia from fluid or pus in alveoli, disrupting airflow. Heard on auscultation, they’re a classic sign, reflecting consolidation or inflammation in affected lung regions.
Choice D reason: Edema is fluid in tissues, linked to heart failure, not pneumonia directly. Pneumonia affects lungs, causing crackles, not peripheral swelling, making this unrelated to typical respiratory findings.
Correct Answer is ["B","D","E"]
Explanation
Choice A reason: Bradypnea (slow breathing) isn’t typical in asthma exacerbation; tachypnea occurs due to airway obstruction. Reduced respiratory rate suggests fatigue or resolution, not active exacerbation, contradicting the physiology of acute bronchoconstriction.
Choice B reason: Wheezing results from turbulent airflow through narrowed bronchioles in asthma exacerbation. It’s a hallmark sign, reflecting reversible obstruction from inflammation and bronchospasm, consistently present during acute episodes requiring intervention.
Choice C reason: Bradycardia isn’t associated with asthma exacerbation; tachycardia is common from hypoxia and stress. Slow heart rate may indicate severe hypoxia late-stage, but it’s not a typical finding in active exacerbation.
Choice D reason: Accessory muscle use (e.g., sternocleidomastoid) compensates for obstructed airflow in asthma exacerbation. It reflects increased work of breathing due to bronchoconstriction, a frequent physical sign during acute respiratory distress.
Choice E reason: Decreased oxygen saturation occurs in asthma exacerbation from ventilation-perfusion mismatch. Inflamed airways limit oxygen delivery, dropping SpO2 below 95%, a common finding necessitating bronchodilators and oxygen therapy.
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