A 78-kg patient in septic shock has a pulse rate of 120 beats/min with low central venous pressure and pulmonary artery wedge pressure. After initial fluid volume resuscitation, the patient's urine output has been 30 mL/hr for the past 3 hours. Which order by the health care provider should the nurse question?
Give hydrocortisone (Solu-Cortef) 100 mg IV.
Administer furosemide (Lasix) 40 mg IV.
Increase normal saline infusion to 250 ml/hr.
Use norepinephrine to keep systolic BP above 90 mm Hg.
The Correct Answer is B
B. Administering furosemide, a loop diuretic, to a patient in septic shock with low urine output could exacerbate hypovolemia and worsen organ perfusion. It could further reduce preload, exacerbating hypotension, and impairing cardiac output. Loop diuretics are typically contraindicated in hypovolemic shock.
A. Corticosteroids such as hydrocortisone can help improve vascular tone and responsiveness to vasopressors. Given the patient's clinical presentation and lack of response to initial fluid resuscitation, administering hydrocortisone is appropriate.
C. Increasing the fluid infusion rate can help restore intravascular volume and improve organ perfusion. However, caution should be exercised to avoid fluid overload and pulmonary edema.
D .Norepinephrine is a vasopressor commonly used to increase systemic vascular resistance and improve blood pressure in septic shock. It helps maintain perfusion pressure to vital organs, thereby supporting organ function.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["A","C","D","E"]
Explanation
A. Hypotension may result from decreased cardiac output due to hypoxemia and acidosis or as a compensatory response to hypoxia-induced vasodilation. Therefore, monitoring for hypotension is important in ARF.
C. Hypoxemia can impair cerebral perfusion and neuronal function, leading to alterations in mental status ranging from confusion to coma. Decreased level of consciousness is a concerning sign of inadequate oxygenation and should be closely monitored in patients with ARF.
D. Dyspnea, or difficulty breathing, is a hallmark symptom of respiratory failure. In ARF, the respiratory system's inability to adequately oxygenate or ventilate leads to increased work of breathing and feelings of breathlessness. Patients with ARF often experience severe dyspnea as they struggle to maintain adequate gas exchange.
E. Headache is not typically considered a primary manifestation of ARF. However, it may occur as a secondary symptom due to factors such as hypoxemia, hypercapnia, or acidosis. Patients with ARF may experience headache as a result of cerebral vasodilation in response to hypoxia or as a symptom of underlying conditions contributing to respiratory failure.
B. Nausea is not a typical manifestation of ARF itself. However, it may occur as a secondary symptom due to factors such as hypoxia, acidosis, or medications administered for the management of ARF. While nausea may be present, it is not a direct result of respiratory failure.
Correct Answer is ["B","C"]
Explanation
B. stroke volume is the amount of blood ejected from the left ventricle with each contraction (systole) of the heart. Changes in stroke volume directly affect cardiac output. An increase in stroke volume leads to an increase in cardiac output, while a decrease in stroke volume results in a decrease in cardiac output. Factors that can affect stroke volume include preload, afterload, and contractility of the heart.
C. Heart rate refers to the number of heartbeats per minute. Heart rate directly affects cardiac output by determining how frequently the heart contracts and pumps blood. An increase in heart rate (tachycardia) leads to an increase in cardiac output, while a decrease in heart rate (bradycardia) results in a decrease in cardiac output. Factors such as sympathetic and parasympathetic nervous system activity, hormones, and medications can influence heart rate.
A. Respiratory rate does not directly affect stroke volume or heart rate but changes in respiratory rate can indirectly impact cardiac output through their effects on venous return and preload.
D. Blood pressure represents the force exerted by the blood against the walls of the arteries. While blood pressure does not directly affect cardiac output, it is influenced by cardiac output and systemic vascular resistance (SVR).
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