A client has been admitted repeatedly in the past year for episodes of atrial flutter which the cardiologist believes are caused by an area of conduction in the atria other than the SA node. Medications have not improved the condition. Which procedure should the nurse anticipate will be performed?
Heart transplantation
Defibrillation
Coronary artery stent placement
Radiofrequency catheter ablation
The Correct Answer is D
A. Heart transplantation is not indicated for atrial flutter; it is reserved for end-stage heart failure or severe cardiac conditions that cannot be managed by other means.
B. Defibrillation is used in emergency situations for life-threatening arrhythmias like ventricular fibrillation, not for atrial flutter.
C. Coronary artery stent placement is used to open blocked coronary arteries, not to treat arrhythmias.
D. Radiofrequency catheter ablation targets and destroys the abnormal conduction pathways causing atrial flutter, providing a more permanent solution when medications are ineffective.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. Lisinopril is not used to reduce LDL cholesterol and triglycerides; statins are used for that purpose.
B. Lisinopril does not prevent platelet aggregation; antiplatelet agents like aspirin are used for that purpose.
C. Lisinopril is not used to relieve acute angina; nitrates like nitroglycerin are used for that purpose.
D. Lisinopril is an ACE inhibitor that helps decrease blood pressure and reduce the workload on the heart, which is beneficial in managing chronic stable angina.
Correct Answer is D
Explanation
A. Romiplostim does not increase the production of ADAMTS-13; it is used to increase platelet counts.
B. Romiplostim does not prevent platelet destruction in the spleen; it works by stimulating platelet production.
C. Romiplostim does not prevent platelets from binding with heparin; it is not used for heparin-induced thrombocytopenia.
D. Romiplostim is a thrombopoietin receptor agonist that stimulates the bone marrow to increase platelet production, which is its primary mechanism of action in treating ITP.
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