A client is receiving (Pitocin) oxytocin to induce labor. The nurse should monitor for which of the following adverse maternal effects?
Jaundice
Dehydration
Uterine hyperstimulation.
Maternal bradycardia
The Correct Answer is C
A. Jaundice. Oxytocin does not cause jaundice in the mother. Jaundice is typically related to liver dysfunction, hemolysis, or bile obstruction and is not an expected side effect of oxytocin administration.
B. Dehydration. While prolonged labor induction may contribute to dehydration if fluid intake is insufficient, oxytocin itself does not directly cause dehydration. IV fluids are typically administered alongside oxytocin to maintain hydration during labor.
C. Uterine hyperstimulation. Uterine hyperstimulation (tachysystole), defined as more than five contractions in 10 minutes, is a serious complication of oxytocin administration. It can lead to fetal distress, uterine rupture, and impaired placental perfusion. If hyperstimulation occurs, the nurse should discontinue oxytocin, reposition the patient, provide oxygen, and notify the provider.
D. Maternal bradycardia. Oxytocin does not typically cause bradycardia. Maternal side effects are more commonly tachycardia, hypertension, or fluid retention. Fetal bradycardia, however, can occur if uterine hyperstimulation leads to fetal hypoxia.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
True.
When supporting a woman who has just experienced a fetal demise, it is appropriate and compassionate to:
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Offer supportive care.
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Allow and encourage space and time to grieve.
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Offer the services of a chaplain, especially if the patient expresses a desire for spiritual or emotional support.
Chaplains are typically trained to be non-denominational and sensitive to a wide range of beliefs, and they can provide comforting words and presence regardless of the patient’s faith or lack thereof. This approach aligns with holistic and patient-centered care.
Correct Answer is C
Explanation
A. Consuming more calories covers the insulin secreted by the fetus. The fetus does not secrete insulin to regulate maternal glucose levels. Instead, the maternal pancreas produces insulin in response to blood sugar levels, but in diabetes, maternal insulin resistance leads to excess glucose being transferred to the fetus.
B. Fetal weight gain increases as a result of the common response of maternal overeating. While some women with diabetes may have increased caloric intake, this is not the primary reason for fetal macrosomia (large birth weight). The major factor is maternal hyperglycemia leading to excess fetal insulin production and fat deposition.
C. Extra circulating glucose causes the fetus to acquire fatty deposits. In diabetic pregnancies, excess maternal glucose crosses the placenta, leading to fetal hyperinsulinemia. The increased insulin promotes fat storage and excessive fetal growth, leading to macrosomia, which increases the risk of birth complications such as shoulder dystocia.
D. Taking exogenous insulin stimulates fetal growth. Insulin does not cross the placenta, so maternal insulin therapy does not directly affect fetal growth. Instead, fetal macrosomia results from prolonged exposure to maternal hyperglycemia, which causes the fetus to produce excessive insulin and store extra fat.
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