A multiparous client, 28 hours after a cesarean birth, who is breastfeeding, has severe cramps or afterpains.
The nurse explains that these are caused by:

Rationale for Correct Condition

Preeclampsia is characterized by hypertension (≥140/90 mm Hg) and proteinuria (≥300 mg/24 hr) or signs of systemic involvement such as headache and edema. The client’s elevated BP, significant proteinuria, headache, hyperreflexia, and pitting edema strongly indicate preeclampsia. The absence of seizures rules out eclampsia, and the gestational age excludes chronic hypertension with superimposed preeclampsia.

Rationale for Correct Actions

Magnesium sulfate prevents seizures by stabilizing neuronal excitability and reducing cerebral vasospasm. It also lowers BP by promoting vascular relaxation. External fetal monitoring detects fetal distress from utero-placental insufficiency caused by preeclampsia-related hypertension and endothelial dysfunction.

Rationale for Correct Parameters

Blood pressure reflects disease progression and response to antihypertensives. Severe hypertension increases the risk of cerebral hemorrhage and placental abruption. Deep tendon reflexes indicate neurological impairment. Hyperreflexia signals worsening preeclampsia, while absent reflexes may indicate magnesium toxicity.

Rationale for Incorrect Conditions

Gestational hypertension lacks proteinuria and systemic symptoms. Chronic hypertension with superimposed preeclampsia requires a pre-existing hypertensive diagnosis. Eclampsia involves seizures, which the client has not reported.

Rationale for Incorrect Actions

Ampicillin is not indicated without infection. Rh immune globulin is unnecessary without Rh incompatibility. Anticoagulants are not first-line for preeclampsia.

Rationale for Incorrect Parameters

Temperature is not directly related to preeclampsia. Petechiae are signs of coagulopathy, not primary indicators. Pruritus is irrelevant unless liver dysfunction is suspected.

Take-Home Points

• Preeclampsia involves multisystem dysfunction with endothelial damage.
• Magnesium sulfate prevents seizures but must be monitored for toxicity.
• Differentiation from gestational hypertension is crucial for management.
• Fetal monitoring is key to detecting early signs of compromise.

Choice A rationale

Hyperemesis gravidarum is severe nausea and vomiting during pregnancy, typically occurring in the first trimester. While amniocentesis can cause some discomfort, it is not a known risk factor for hyperemesis.

Choice B rationale

Proteinuria is the presence of protein in the urine, which can be a sign of preeclampsia, a condition that can occur later in pregnancy. Amniocentesis, usually performed in the second trimester, is not directly linked to causing proteinuria.

Choice C rationale

Amniocentesis is an invasive procedure that involves inserting a needle through the abdominal wall and uterus to withdraw amniotic fluid. This procedure carries a risk of bleeding or hemorrhage from the insertion site or within the uterus. Therefore, observing for signs of hemorrhage is a crucial nursing intervention post-amniocentesis.

Choice D rationale

Hypoxia is a condition of insufficient oxygen supply to the tissues. While complications of amniocentesis could indirectly lead to fetal hypoxia in rare cases (e.g., placental abruption), it is not a direct and common complication to observe for in the mother. Hemorrhage is a more immediate and direct risk for the mother. .