A nurse and a newly licensed nurse are providing care for a client who has distributive shock. How should the nurse explain the pathophysiology of distributive shock to the newly licensed nurse?
"Distributive shock occurs due to loss of myocardial contractility."
"Distributive shock occurs due to loss of blood volume."
"Distributive shock occurs due to systemic vasodilation."
"Distributive shock occurs due to increased systemic vascular resistance."
The Correct Answer is C
A. "Distributive shock occurs due to loss of myocardial contractility": This statement is incorrect. Distributive shock is not primarily caused by loss of myocardial contractility. Instead, distributive shock is characterized by widespread vasodilation, which leads to inadequate tissue perfusion despite normal or high cardiac output.
B. "Distributive shock occurs due to loss of blood volume": This statement is inaccurate. Distributive shock is not primarily caused by loss of blood volume. While hypovolemia (loss of blood volume) can lead to shock, distributive shock specifically involves excessive vasodilation, resulting in a relative hypovolemia due to pooling of blood in the expanded vascular bed.
C. "Distributive shock occurs due to systemic vasodilation": This statement is correct. Distributive shock, also known as vasodilatory shock, occurs due to widespread vasodilation of the systemic vasculature. This vasodilation leads to a decrease in systemic vascular resistance, which results in the redistribution of blood flow away from vital organs and tissues, leading to inadequate tissue perfusion and shock.
D. "Distributive shock occurs due to increased systemic vascular resistance": This statement is incorrect. Distributive shock is characterized by decreased systemic vascular resistance due to vasodilation, not increased systemic vascular resistance. Increased systemic vascular resistance is more commonly associated with conditions such as hypertension or obstructive shock.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is D
Explanation
A. "I should ask my son to drive me to the grocery store." This statement does not directly address the risk for sudden cardiac death associated with arrhythmogenic cardiomyopathy. While it may be a valid safety precaution to have someone else drive, it does not specifically address the client's understanding of the condition and its implications for sudden cardiac death.
B. "I will probably become easily constipated." Constipation is not a typical manifestation or complication of arrhythmogenic cardiomyopathy. This statement does not demonstrate an understanding of the condition and its associated risk of sudden cardiac death.
C. "I am aware that I may develop frequent hiccups." Frequent hiccups are not a characteristic symptom or complication of arrhythmogenic cardiomyopathy. This statement does not indicate an understanding of the condition and its risk for sudden cardiac death.
D. "I will need to avoid strenuous activity to prevent my heart from stopping." This statement reflects an understanding of the condition and its associated risk of sudden cardiac death. Strenuous activity can exacerbate arrhythmias in individuals with arrhythmogenic cardiomyopathy, potentially leading to life-threatening arrhythmias such as ventricular fibrillation. Avoiding strenuous activity is a recommended precaution to reduce the risk of sudden cardiac death in individuals with this condition.
Correct Answer is B
Explanation
A. Poor functional ability: While poor functional ability may impact the overall prognosis and quality of life for a client with a subarachnoid hemorrhage (SAH), it is not directly associated with a high mortality rate. Functional ability can be improved with rehabilitation and supportive care.
B. Rebleeding of the injury: Rebleeding of the SAH is a significant risk factor associated with a high mortality rate. Rebleeding can lead to increased intracranial pressure, worsening neurological deficits, and even death. Preventing rebleeding is a critical aspect of managing SAH to improve outcomes.
C. Decreased cerebrospinal fluid: Decreased cerebrospinal fluid (CSF) may indicate conditions such as hydrocephalus, which can complicate the management of SAH. However, it is not directly associated with a high mortality rate compared to rebleeding.
D. Use of nimodipine: Nimodipine is a calcium channel blocker commonly used in the management of SAH to prevent cerebral vasospasm, which can lead to ischemia and worsen outcomes. While nimodipine plays a role in improving outcomes by preventing vasospasm, its use is not directly associated with mortality rates.
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