A nurse and a newly licensed nurse are providing care for a client who has distributive shock. How should the nurse explain the pathophysiology of distributive shock to the newly licensed nurse?
"Distributive shock occurs due to loss of myocardial contractility."
"Distributive shock occurs due to loss of blood volume."
"Distributive shock occurs due to systemic vasodilation."
"Distributive shock occurs due to increased systemic vascular resistance."
The Correct Answer is C
A. "Distributive shock occurs due to loss of myocardial contractility": This statement is incorrect. Distributive shock is not primarily caused by loss of myocardial contractility. Instead, distributive shock is characterized by widespread vasodilation, which leads to inadequate tissue perfusion despite normal or high cardiac output.
B. "Distributive shock occurs due to loss of blood volume": This statement is inaccurate. Distributive shock is not primarily caused by loss of blood volume. While hypovolemia (loss of blood volume) can lead to shock, distributive shock specifically involves excessive vasodilation, resulting in a relative hypovolemia due to pooling of blood in the expanded vascular bed.
C. "Distributive shock occurs due to systemic vasodilation": This statement is correct. Distributive shock, also known as vasodilatory shock, occurs due to widespread vasodilation of the systemic vasculature. This vasodilation leads to a decrease in systemic vascular resistance, which results in the redistribution of blood flow away from vital organs and tissues, leading to inadequate tissue perfusion and shock.
D. "Distributive shock occurs due to increased systemic vascular resistance": This statement is incorrect. Distributive shock is characterized by decreased systemic vascular resistance due to vasodilation, not increased systemic vascular resistance. Increased systemic vascular resistance is more commonly associated with conditions such as hypertension or obstructive shock.
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Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is ["B","C","D"]
Explanation
Pulseless electrical activity (PEA) refers to a situation in which an electrocardiogram (ECG) shows electrical activity in the heart, but there is no effective mechanical activity and no palpable pulse. PEA can result from various underlying conditions that impair cardiac function or compromise circulation.
A. Hypertension: Hypertension, or high blood pressure, is not typically a direct cause of PEA. While uncontrolled hypertension can contribute to cardiovascular disease and increase the risk of cardiac events such as myocardial infarction, it is not a direct cause of PEA.
B. Hypovolemia: Hypovolemia, or low blood volume, is a common cause of PEA. Reduced circulating blood volume leads to decreased preload and inadequate filling of the heart chambers, impairing cardiac output. This can result in ineffective cardiac contractions and the development of PEA.
C. Hypoxia: Hypoxia, or inadequate oxygenation of tissues, is a significant cause of PEA. Insufficient oxygen delivery to the myocardium impairs cardiac function and can lead to ineffective cardiac contractions. Without adequate oxygenation, the heart is unable to generate sufficient mechanical force to maintain circulation, resulting in PEA.
D. Hyperkalemia: Hyperkalemia, or elevated levels of potassium in the blood, can cause PEA by disrupting cardiac electrical activity. Excessive potassium ions in the extracellular fluid alter the normal myocardial action potential, leading to cardiac conduction abnormalities and potential arrhythmias, including PEA.
E. Hypernatremia: Hypernatremia, or elevated levels of sodium in the blood, is not typically a direct cause of PEA. While severe hypernatremia can lead to neurological symptoms and dehydration, it does not directly impair cardiac electrical activity or mechanical function to the extent that it causes PEA.
F. Hyperthermia: Hyperthermia, or elevated body temperature, is not typically a direct cause of PEA. While extreme hyperthermia can lead to cardiovascular complications such as heat stroke, it is not a common cause of PEA unless it is associated with severe systemic metabolic derangements.
Correct Answer is C
Explanation
A. "Since my parent suffered from Alzheimer's disease, I know that I am at an increased risk for developing the disease myself." This statement is accurate. Family history is a significant risk factor for Alzheimer's disease. Individuals with a first-degree relative (such as a parent or sibling) with Alzheimer's disease are at a higher risk of developing the condition themselves.
B. "The cause of Alzheimer's disease is still not fully known or understood." This statement is also accurate. While there are theories about the underlying causes of Alzheimer's disease, such as genetics, brain changes, and environmental factors, the exact cause is still not fully understood. Research into the etiology of Alzheimer's disease is ongoing.
C. "I do not have to worry about this because I do not have Down syndrome and I have never had a stroke." This statement indicates a need for further teaching. While it is true that individuals with Down syndrome and those who have had a stroke are at increased risk for developing Alzheimer's disease, they are not the only populations at risk. Alzheimer's disease can affect individuals without Down syndrome or a history of stroke. Other risk factors include age, family history, genetics, and lifestyle factors.
D. "My child is at risk for developing Alzheimer's disease because they have trisomy 21." This statement is accurate. Trisomy 21, also known as Down syndrome, is associated with an increased risk of developing Alzheimer's disease. Individuals with Down syndrome have three copies of chromosome 21, which contains the gene for amyloid precursor protein (APP). Overproduction of amyloid beta protein, derived from APP, is thought to contribute to the development of Alzheimer's disease in individuals with Down syndrome.
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