A nurse is caring for a client in the intensive care unit. Which of the following laboratory values could contribute to an episode of delirium?
White blood cell level of 5,900 mm3
Potassium level of 4.1 mEq/L
Hemoglobin level of 14.2 g/dL
Blood glucose level of 254 mg/dL
The Correct Answer is D
A. White blood cell level of 5,900 mm3: While abnormal white blood cell levels can indicate infection or inflammation, they are not typically associated with directly contributing to an episode of delirium. However, underlying conditions that cause abnormal white blood cell levels, such as infection or inflammation, may contribute to delirium.
B. Potassium level of 4.1 mEq/L: Potassium imbalances can lead to various neurological symptoms, including weakness, paralysis, and cardiac arrhythmias. However, a potassium level of 4.1 mEq/L is within the normal range and is unlikely to directly contribute to an episode of delirium.
C. Hemoglobin level of 14.2 g/dL: Hemoglobin levels reflect the oxygen-carrying capacity of the blood and are not directly associated with delirium. While severe anemia or hypoxia can cause neurological symptoms, a hemoglobin level of 14.2 g/dL is within the normal range and is unlikely to directly contribute to delirium.
D. Blood glucose level of 254 mg/dL: Elevated blood glucose levels, as indicated by a blood glucose level of 254 mg/dL, can contribute to an episode of delirium. Hyperglycemia can lead to alterations in cerebral metabolism, neuronal dysfunction, and impaired cognitive function, predisposing individuals to delirium. Additionally, hyperglycemia can exacerbate preexisting neurological conditions and increase the risk of developing delirium in critically ill patients. Therefore, monitoring and managing blood glucose levels are essential in preventing and managing delirium in hospitalized patients.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
A. The heart's electrical transmission through the atrioventricular (AV) node is unusually slow: This statement does not align with the manifestations of dizziness and palpitations in atrial fibrillation. A slow conduction through the AV node would typically manifest as bradycardia or heart block rather than rapid and irregular palpitations.
B. The heart's electrical signals are rapid, chaotic, and irregular: This is the correct answer. Atrial fibrillation is characterized by rapid, chaotic, and irregular electrical signals in the atria, leading to ineffective atrial contractions and an irregular ventricular response. The irregularity and rapidity of the heart rate can lead to symptoms such as palpitations and dizziness.
C. An early electrical signal occurs before the expected sinoatrial (SA) node signal: This statement does not accurately describe the mechanism of atrial fibrillation. Atrial fibrillation involves disorganized electrical activity in the atria, rather than a single early electrical signal.
D. The SA node sends an electrical signal greater than 100/min: While a heart rate greater than 100/min (tachycardia) is commonly associated with atrial fibrillation, this statement does not fully explain the manifestations of dizziness and palpitations. These symptoms are more directly related to the irregularity and chaotic nature of the heart's electrical signals in atrial fibrillation.
Correct Answer is ["D","E"]
Explanation
A. Diabetes mellitus: An inverse association between diabetes mellitus (DM) and abdominal aortic aneurysm (AAA) risk hasbeen reported. Apart from a lower AAA prevalence among patients with vsthose without DM, there isdata showing that DM may exert a protective role on aneurysmal growth in patients with small AAAs, thus decreasing the risk of rupture. As atherosclerosis has almost the same risk factors as aneurysms, the decreased AAA prevalence in patients with DM may indicate that atherosclerosis is an associated feature and not a cause of the aneurysms.
B. Total cholesterol 170 mg/dL (less than 200 mg/dL): While elevated total cholesterol is a risk factor for cardiovascular disease, it is not specifically listed as a risk factor for abdominal aortic aneurysm (AAA). However, dyslipidemia, including elevated total cholesterol levels, can contribute to the development of atherosclerosis, which is a risk factor for AAA.
C. HDL cholesterol 65 mg/dL (male greater than 45 mg/dL; female greater than 55 mg/dL): High-density lipoprotein (HDL) cholesterol levels greater than 65 mg/dL are not listed as a risk factor for abdominal aortic aneurysm (AAA). However, low levels of HDL cholesterol are associated with an increased risk of cardiovascular disease, which may indirectly contribute to the development of AAA through the promotion of atherosclerosis.
D. Smoking cigarettes: Smoking cigarettes is a significant modifiable risk factor for abdominal aortic aneurysm (AAA). Smoking damages the walls of blood vessels, promotes inflammation, and accelerates the development of atherosclerosis, increasing the risk of AAA formation and rupture.
E. Family history of aneurysm: A family history of aneurysm, particularly abdominal aortic aneurysm (AAA), is a risk factor for developing AAA. Genetic factors can predispose individuals to the development of aneurysms, and a positive family history increases the likelihood of AAA occurrence.
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