A nurse is collecting neurologic data on a client who has a neurological injury and notes changes suggestive of Cushing's Triad. The nurse understands that Cushing's Triad is a nervous system response that might prevent which of the following conditions?

A. Elevated erythrocyte sedimentation rate (ESR): Elevated ESR is commonly seen in inflammatory conditions such as pericarditis. ESR measures the rate at which red blood cells settle in a tube of blood over a certain period, and elevated levels indicate inflammation or tissue damage.

B. Increased C-reactive protein (CRP): CRP is an acute-phase reactant produced by the liver in response to inflammation. Elevated CRP levels are indicative of inflammation, making it a useful marker in pericarditis.

C. Elevated thyroid-stimulating hormone (TSH): Elevated TSH levels are not typically associated with pericarditis. TSH is a hormone produced by the pituitary gland that stimulates the thyroid gland to produce thyroid hormones. Elevated TSH levels are seen in conditions such as hypothyroidism.

D. Increased brain natriuretic peptide (BNP): BNP is a hormone produced by the heart in response to increased pressure and volume overload. Pericarditis can lead to increased pressure within the heart, resulting in elevated BNP levels. Therefore, increased BNP levels are anticipated in pericarditis.

E. Increased troponin I: Troponin I is a cardiac biomarker released into the bloodstream when there is damage to cardiac muscle cells. While pericarditis primarily involves inflammation of the pericardium (the sac surrounding the heart), severe cases can lead to myocardial involvement and subsequent release of troponin I. Therefore, increased troponin I levels may be observed in pericarditis, especially if there is myocardial involvement.

A. "Your heart condition is caused by excessive stretching of the ventricles": Dilated cardiomyopathy (DCM) is characterized by the dilation or enlargement of the heart's chambers, particularly the ventricles. This dilation results in weakened myocardial contraction and impaired systolic function. The primary cause of DCM is often idiopathic (unknown), but it can also be secondary to various factors such as genetics, infections, toxins, or systemic diseases. The statement correctly identifies the pathophysiological basis of DCM as excessive stretching or dilation of the ventricles, leading to impaired cardiac function.

B. "Your heart condition is caused when the ventricular tissue becomes fibrous and fatty": This statement is incorrect. Fibrous and fatty changes in ventricular tissue are characteristic of another type of cardiomyopathy known as hypertrophic cardiomyopathy (HCM), not dilated cardiomyopathy (DCM). In DCM, the primary abnormality is dilation of the heart chambers rather than fibrous and fatty changes in the myocardium.

C. "Your heart condition is caused from stiffening of the walls of the ventricles": This statement describes restrictive cardiomyopathy (RCM), not dilated cardiomyopathy (DCM). In restrictive cardiomyopathy, the ventricular walls become stiff and rigid, impairing diastolic filling and ventricular relaxation. However, in DCM, the primary abnormality is dilation or enlargement of the ventricles rather than stiffening of the ventricular walls.

D. "Your heart condition is caused by thickening of the ventricular walls and septum": This statement describes hypertrophic cardiomyopathy (HCM), not dilated cardiomyopathy (DCM). In HCM, there is abnormal thickening of the ventricular walls and septum, leading to reduced chamber size and impaired diastolic filling. However, in DCM, the primary abnormality is dilation or enlargement of the ventricles rather than thickening of the ventricular walls.