During lunch, a patient cuts herself with a plastic eating utensil. The patient refuses redirection and continues to attempt to cut herself. The patient is safely secured in a restraint chair. What is the next priority action for the nurse?
Notify the provider for a restraint order and assessment.
Advise the cafeteria staff of plastic utensils that can be used as weapons.
Ask staff members how the patient was able to acquire the utensil.
Notify the house supervisor for further direction.
The Correct Answer is A
Choice A reason: Notifying the provider ensures legal and medical oversight for restraints, addressing self-harm risk driven by serotonin deficits and amygdala hyperactivity. This prioritizes patient safety and evaluates underlying psychiatric causes, ensuring appropriate intervention to stabilize neural dysregulation.
Choice B reason: Advising cafeteria staff is a preventive measure but not the priority during acute self-harm. Serotonin-driven impulsivity requires immediate medical assessment, and addressing utensil access is secondary to stabilizing the patient’s acute neurochemical crisis.
Choice C reason: Investigating utensil acquisition is important for future prevention but not urgent. The patient’s self-harm, driven by serotonin deficits and amygdala hyperactivity, requires immediate medical intervention to ensure safety and address acute psychiatric needs first.
Choice D reason: Notifying the house supervisor is administrative, not clinical, and delays direct intervention. Self-harm reflects serotonin dysregulation and amygdala-driven impulsivity, requiring immediate provider assessment for restraints and psychiatric evaluation, making this less urgent.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is B
Explanation
Choice A reason: Olanzapine does not primarily increase serotonin or norepinephrine. It blocks dopamine D2 and serotonin 5-HT2 receptors, reducing hallucinations driven by mesolimbic dopamine excess in schizophrenia. Increasing monoamines would exacerbate psychosis, not alleviate it, making this mechanism incorrect.
Choice B reason: Olanzapine, an atypical antipsychotic, blocks dopamine D2 receptors in the mesolimbic pathway, reducing excessive dopamine signaling that causes hallucinations in schizophrenia. It also modulates serotonin 5-HT2 receptors, balancing limbic activity, making this the primary mechanism for controlling psychotic symptoms.
Choice C reason: Decreasing neurotransmitter breakdown enzymes is the mechanism of MAOIs, not olanzapine. Olanzapine’s dopamine and serotonin receptor blockade reduces psychotic symptoms, not enzyme activity, making this choice irrelevant to its antipsychotic action in schizophrenia.
Choice D reason: Olanzapine does not normalize serotonin, norepinephrine, and dopamine levels but blocks their receptors, particularly dopamine D2, to reduce hallucinations. Normalizing levels is not its mechanism, as schizophrenia involves dopamine hyperactivity, not deficiency, making this choice inaccurate.
Correct Answer is C
Explanation
Choice A reason: Attachment-Based Therapy focuses on interpersonal relationships, not trauma-specific processing, and is less effective for PTSD’s amygdala-driven fear memories. Mirtazapine enhances serotonin and norepinephrine but lacks strong evidence for PTSD, as it primarily addresses depression by modulating alpha-2 adrenergic receptors, not trauma-specific neural hyperactivity.
Choice B reason: Dialectical Behavior Therapy targets emotional regulation for personality disorders, not PTSD’s trauma-specific memories. Lorazepam, a benzodiazepine, enhances GABA activity but is not recommended for PTSD, as it may suppress rather than process trauma-related amygdala hyperactivity, risking dependency and impairing long-term recovery.
Choice C reason: Cognitive Processing Therapy restructures trauma-related cognitive distortions, reducing amygdala hyperactivity and enhancing prefrontal control in PTSD. Sertraline, an SSRI, increases serotonin levels, stabilizing mood and reducing hyperarousal by modulating amygdala-prefrontal circuits, making it a first-line treatment for PTSD’s neurochemical and cognitive symptoms.
Choice D reason: Applied Behavior Analysis is used for autism, not PTSD, and does not address trauma-related neural changes. Atomoxetine, a norepinephrine reuptake inhibitor, targets ADHD by enhancing prefrontal norepinephrine but lacks efficacy for PTSD’s amygdala-driven fear responses, making it unsuitable for trauma treatment.
Whether you are a student looking to ace your exams or a practicing nurse seeking to enhance your expertise , our nursing education contents will empower you with the confidence and competence to make a difference in the lives of patients and become a respected leader in the healthcare field.
Visit Naxlex, invest in your future and unlock endless possibilities with our unparalleled nursing education contents today
Report Wrong Answer on the Current Question
Do you disagree with the answer? If yes, what is your expected answer? Explain.
Kindly be descriptive with the issue you are facing.