The nurse is caring for a patient in the Intensive Care Unit (ICU) with a ventriculostomy and intracranial pressure (ICP) reading of 30mmHg. The nurse notes a widened pulse pressure, irregular respirations. and bradycardia. The neurosurgeon instructs the nurse to drain cerebrospinal fluid. The nurse would also anticipate which medication administration order from the provider?
3% Normal Saline
Nitroprusside
Norepinephrine
Furosemide
The Correct Answer is A
A) 3% Normal Saline:
Elevated ICP can lead to a life-threatening condition known as cerebral herniation, and it often triggers the Cushing’s triad—widened pulse pressure, bradycardia, and irregular respirations, all of which the nurse has noted. Hypertonic saline (such as 3% Normal Saline) is commonly used to manage increased ICP because it creates an osmotic gradient that draws fluid out of the brain tissue, reducing cerebral edema and lowering ICP. The goal is to reduce swelling and prevent further brain damage.
B) Nitroprusside:
Nitroprusside is a potent vasodilator that is used to treat hypertension and reduce afterload in conditions like heart failure or aortic dissection. However, in this case, nitroprusside is contraindicated because it could cause a drop in systemic blood pressure, which would worsen cerebral perfusion in a patient with elevated ICP.
C) Norepinephrine:
Norepinephrine is a vasopressor used to treat hypotension by increasing systemic vascular resistance and blood pressure. Although norepinephrine may be used to support blood pressure in critically ill patients, in this context of elevated ICP, the priority is to reduce ICP, not to increase blood pressure further. Increasing the systemic vascular resistance with norepinephrine could exacerbate ICP by increasing the volume of blood in the cranium
D) Furosemide:
Furosemide is a loop diuretic that can reduce fluid volume and help treat conditions like edema or heart failure. While furosemide can reduce overall fluid volume, it is not typically used to manage elevated ICP in this context. Diuretics like furosemide may have a role in managing cerebral edema, but they are not as effective as hypertonic saline in rapidly reducing ICP.
Nursing Test Bank
Naxlex Comprehensive Predictor Exams
Related Questions
Correct Answer is A
Explanation
A) Assess lung sounds and respiratory rate at least every 2 hours:
In a patient with Myasthenia Gravis (MG) who has undergone thymectomy, monitoring respiratory status is critical. MG is a neuromuscular disorder that can lead to respiratory muscle weakness, which may be exacerbated post-operatively. Assessing lung sounds and respiratory rate at least every 2 hours is crucial to detect early signs of respiratory compromise, including hypoventilation or atelectasis.
B) Assess and document pain level once every shift:
While pain assessment is important, especially after a thymectomy, this action alone does not directly address the immediate issue of ineffective breathing patterns. In patients with MG, respiratory complications are a priority concern. Pain management should be part of the overall plan of care, but it is secondary to monitoring respiratory function in the acute post-operative period. Pain can affect respiratory effort, but it should be managed in the context of more pressing issues like airway and breathing assessment.
C) Maintain sequential compression device (SCD's) while in bed:
While SCDs are important in preventing deep vein thrombosis (DVT) in patients who are immobile, they are not the most appropriate intervention for a client with ineffective breathing patterns. The primary concern in a post-thymectomy patient with MG is respiratory function.
D) Elevate the head of the bed ten degrees:
While elevating the head of the bed can help with comfort and potentially improve ventilation in some patients, it is not the most specific or effective intervention for managing ineffective breathing patterns in a patient with MG. For optimal respiratory function, it is typically more beneficial to elevate the head of the bed to a higher degree (e.g., 30-45 degrees) to enhance lung expansion, rather than just 10 degrees.
Correct Answer is ["A","B","D"]
Explanation
A) Tachycardia: Tachycardia is commonly seen in left-sided heart failure as the heart attempts to compensate for the reduced cardiac output. To maintain adequate perfusion, the body increases the heart rate. The sympathetic nervous system is activated, causing an increase in heart rate to try to pump blood more efficiently despite the reduced pumping ability of the left ventricle.
B) Crackles: Crackles (also known as rales) are typically heard upon auscultation of the lungs in patients with left-sided heart failure. When the left ventricle fails to effectively pump blood, it causes blood to back up into the lungs, resulting in pulmonary congestion. This leads to fluid accumulation in the alveoli, causing crackling sounds during inhalation.
C) Ascites: Ascites is more commonly seen in right-sided heart failure or in cases of congestive heart failure where both sides of the heart are affected. Ascites is the accumulation of fluid in the abdomen, which is a consequence of right-sided heart failure causing blood to back up into the abdomen. In left-sided heart failure, ascites is generally not a primary symptom unless the failure becomes severe and involves both sides of the heart.
D) Dyspnea: Dyspnea, or shortness of breath, is a hallmark symptom of left-sided heart failure. The left ventricle's inability to pump blood efficiently leads to pulmonary congestion, which causes fluid to accumulate in the lungs. This fluid buildup reduces the lung's ability to exchange oxygen, resulting in difficulty breathing, especially on exertion or when lying down (orthopnea).
E) Peripheral edema: Peripheral edema is more characteristic of right-sided heart failure due to the backup of blood in the systemic circulation. While it can occur in cases of biventricular heart failure (both right and left sides of the heart are affected), it is not the primary finding in left-sided heart failure. Left-sided heart failure typically presents with pulmonary symptoms rather than systemic symptoms like peripheral edema.
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